Stepwise unfolding of human β2-microglobulin into a disordered amyloidogenic precursor at low pH

Dominic Narang1, Anubhuti Singh2, Samrat Mukhopadhyay3,4

  • 1Department of Biological Sciences, Centre for Protein Science, Design and Engineering, Indian Institute of Science Education and Research (IISER), Mohali, Mohali, India.

Insights

Human beta2-microglobulin (β2m) unfolding at low pH reveals a complex, stepwise mechanism. This process generates a disordered amyloid precursor, crucial for understanding dialysis-related amyloidosis.

Area of Science:

  • Biochemistry
  • Structural Biology
  • Protein Misfolding Diseases

Background:

  • Dialysis-related amyloidosis is linked to amyloid fibril formation by human beta2-microglobulin (β2m).
  • Understanding protein unfolding intermediates is key to elucidating misfolding mechanisms.

Purpose of the Study:

  • To investigate the pH-induced unfolding mechanism of β2m.
  • To characterize the structural properties of unfolding intermediates.

Main Methods:

  • Spectroscopic techniques (e.g., fluorescence) were employed to monitor unfolding.
  • Single tryptophan mutants (W60 and W95) were created to probe local structural changes.
  • Stopped-flow kinetics were used to determine unfolding timescales.

Main Results:

  • At pH 2.5, W60 showed non-native local structure, while W95 became more exposed.
  • β2m unfolding proceeds stepwise, involving initial protein expansion and core solvation.
  • A distinct unfolding phase occurred over ~500 ms, transitioning to a disordered state.

Conclusions:

  • β2m exhibits a complex, multi-step pH-induced unfolding pathway.
  • The process generates a disordered, amyloidogenic precursor with mixed structural features.
  • This provides insights into the structural basis of dialysis-related amyloidosis.

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