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Updated: Mar 18, 2026

Methyl-binding DNA capture Sequencing for Patient Tissues
Published on: October 31, 2016
Methylation Landscape of Human Breast Cancer Cells in Response to Dietary Compound Resveratrol
Rubiceli Medina-Aguilar1, Carlos Pérez-Plasencia2, Laurence A Marchat3
1Departamento de Genética y Biología Molecular, CINVESTAV-IPN, Ciudad de México, México.
Abstract:
Aberrant DNA methylation is a frequent epigenetic alteration in cancer cells that has emerged as a pivotal mechanism for tumorigenesis. Accordingly, novel therapies targeting the epigenome are being explored with the aim to restore normal DNA methylation patterns on oncogenes and tumor suppressor genes. A limited number of studies indicate that dietary compound resveratrol modulates DNA methylation of several cancer-related genes; however a complete view of changes in methylome by resveratrol has not been reported yet. In this study we performed a genome-wide survey of DNA methylation signatures in triple negative breast cancer cells exposed to resveratrol. Our data showed that resveratrol treatment for 24 h and 48 h decreased gene promoter hypermethylation and increased DNA hypomethylation. Of 2476 hypermethylated genes in control cells, 1,459 and 1,547 were differentially hypomethylated after 24 h and 48 h, respectively. Remarkably, resveratrol did not induce widespread non-specific DNA hyper- or hypomethylation as changes in methylation were found in only 12.5% of 27,728 CpG loci. Moreover, resveratrol restores the hypomethylated and hypermethylated status of key tumor suppressor genes and oncogenes, respectively. Importantly, the integrative analysis of methylome and transcriptome profiles in response to resveratrol showed that methylation alterations were concordant with changes in mRNA expression. Our findings reveal for the first time the impact of resveratrol on the methylome of breast cancer cells and identify novel potential targets for epigenetic therapy. We propose that resveratrol may be considered as a dietary epidrug as it may exert its anti-tumor activities by modifying the methylation status of cancer -related genes which deserves further in vivo characterization.
Insights
Resveratrol, a dietary compound, modifies DNA methylation in triple-negative breast cancer cells, restoring normal gene methylation patterns. This epigenetic reprogramming by resveratrol offers potential for novel dietary cancer therapies.
Area of Science:
- Epigenetics
- Cancer Biology
- Nutritional Biochemistry
Background:
- Aberrant DNA methylation is a key driver of cancer development.
- Epigenetic therapies aim to correct abnormal methylation patterns in oncogenes and tumor suppressor genes.
- Resveratrol's impact on the cancer methylome is not fully understood.
Purpose of the Study:
- To investigate the genome-wide DNA methylation changes induced by resveratrol in triple-negative breast cancer cells.
- To determine if resveratrol can restore normal methylation patterns in cancer-related genes.
- To explore resveratrol as a potential dietary epigenetic drug.
Main Methods:
- Genome-wide DNA methylation profiling of triple-negative breast cancer cells treated with resveratrol for 24 and 48 hours.
- Analysis of changes in gene promoter hypermethylation and hypomethylation.
- Integrative analysis of methylome and transcriptome data.
Main Results:
- Resveratrol treatment decreased gene promoter hypermethylation and increased DNA hypomethylation.
- Resveratrol selectively altered methylation in 12.5% of CpG loci, not causing widespread non-specific changes.
- Methylation changes were concordant with alterations in mRNA expression, affecting tumor suppressor genes and oncogenes.
Conclusions:
- Resveratrol significantly impacts the methylome of breast cancer cells, selectively reversing aberrant methylation.
- Resveratrol's ability to normalize methylation of key cancer genes suggests potential as a dietary epigenetic therapeutic agent.
- Further in vivo studies are warranted to confirm resveratrol's anti-tumor activities via epigenetic modulation.

