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Increased platelet intracellular calcium concentration in patients with bipolar affective disorders

S L Dubovsky1, J Christiano, L C Daniell

  • 1Department of Psychiatry, University of Colorado School of Medicine, Denver.

Insights

Blood platelets in patients with mania show increased intracellular calcium. Bipolar patients, both manic and depressed, exhibit heightened calcium responses to stimulation, unlike unipolar patients.

Area of Science:

  • Neuroscience
  • Biochemistry
  • Psychiatry

Background:

  • Intracellular calcium ions play a crucial role in platelet activation and signaling.
  • Dysregulation of intracellular calcium has been implicated in various mood disorders.
  • Platelets offer a accessible model for studying cellular mechanisms in psychiatric conditions.

Purpose of the Study:

  • To investigate differences in free intracellular calcium ion concentration in blood platelets across various mood disorder states.
  • To compare calcium mobilization responses to specific agonists between patients with mania, bipolar depression, unipolar depression, and healthy controls.

Main Methods:

  • Utilized the fluorescent indicator Fura 2 to measure free intracellular calcium ion concentration.
  • Assessed baseline calcium levels and responses to platelet-activating factor and thrombin stimulation.
  • Included patients with untreated mania, bipolar depression, unipolar depression, recovered patients, and age/sex-matched controls.

Main Results:

  • Significantly increased baseline intracellular calcium in platelets of patients with mania compared to controls.
  • Significantly higher intracellular calcium after stimulation in platelets of manic and bipolar depressed patients versus other groups.
  • A significantly lower increase in intracellular calcium over baseline upon stimulation in unipolar patients compared to bipolar patients.

Conclusions:

  • Platelets from manic and bipolar depressed patients demonstrate enhanced intracellular calcium activity.
  • Unipolar patients show a distinct impairment in intracellular calcium mobilization in response to stimulation.
  • These findings suggest differential platelet calcium handling mechanisms in bipolar versus unipolar depression.

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