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Related Concept Videos

Chronic Pancreatitis I: Introduction01:24

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The pancreas, an elongated and flat gland situated behind the stomach, serves a vital function in digesting food and managing blood sugar levels.
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Acute pancreatitis presents a complex medical emergency characterized by rapid onset inflammation of the pancreas, demanding timely diagnosis and management to prevent complications. The condition primarily manifests through severe upper abdominal pain that often radiates to the back. This pain intensifies following the consumption of fatty foods. Accompanying symptoms such as nausea, vomiting, abdominal distention, fever, dyspnea, cyanosis, and jaundice can vary in intensity but significantly...
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Sodium Taurocholate Induced Severe Acute Pancreatitis in C57BL/6 Mice
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Hypertriglyceridemic pancreatitis associated with confounding laboratory abnormalities.

Stephen Melnick1, Salik Nazir2, David Gish2

  • 1Department of Internal Medicine, Reading Health System, West Reading, PA, USA; Stephen.Melnick2@readinghealth.org.

Journal of Community Hospital Internal Medicine Perspectives
|July 14, 2016
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Summary

This case study discusses acute pancreatitis caused by severe hypertriglyceridemia. It emphasizes recognizing falsely low triglyceride and amylase levels in such cases for accurate diagnosis and treatment.

Keywords:
epigastric painhypertriglyceridepancreatitis

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Area of Science:

  • Internal Medicine
  • Gastroenterology
  • Clinical Chemistry

Background:

  • Hypertriglyceridemia is a significant risk factor for acute pancreatitis.
  • Accurate laboratory measurement of triglycerides and amylase is crucial for diagnosing hypertriglyceridemic pancreatitis.
  • Pseudohyponatremia can occur in severe hypertriglyceridemia.

Observation:

  • A 36-year-old woman presented with epigastric pain and tenderness.
  • Initial laboratory results showed high lipase, normal amylase, pseudohyponatremia, and a falsely low triglyceride level.
  • Repeat triglyceride measurement revealed a significantly elevated level (>10,000 mg/dl).

Findings:

  • The patient's clinical presentation was consistent with acute pancreatitis secondary to severe hypertriglyceridemia.
  • Initial laboratory values for amylase and triglycerides were misleading due to the underlying condition.
  • Intravenous insulin and plasmapheresis were required for management.

Implications:

  • Clinicians must be aware of potential laboratory result discrepancies in hypertriglyceridemic pancreatitis.
  • Prompt recognition of falsely low triglyceride and amylase levels is vital for appropriate patient management.
  • This case underscores the importance of considering hypertriglyceridemia in unexplained pancreatitis presentations.