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Hypomagnesemia in a Tennis Player.
The Physician and Sportsmedicine
|July 20, 2016
Summary
Severe exercise can trigger carpopedal spasms due to low magnesium levels (Hypomagnesemia). Magnesium supplementation effectively resolved these symptoms in a patient experiencing prolonged physical exertion.
Area of Science:
- Sports Medicine
- Clinical Biochemistry
- Neurology
Background:
- Prolonged and intense physical activity can precipitate various physiological disturbances.
- Carpopedal spasm, characterized by involuntary contractions of hand and foot muscles, can be a symptom of electrolyte imbalances.
Purpose of the Study:
- To report a case of carpopedal spasm associated with severe exercise.
- To investigate the potential link between Hypomagnesemia and exercise-induced spasms.
- To highlight the efficacy of magnesium supplementation in managing these symptoms.
Main Methods:
- Case report detailing a patient with a history of severe, prolonged exercise.
- Clinical assessment including symptom evaluation and serum magnesium level measurement.
- Monitoring of symptoms following magnesium supplementation.
Main Results:
- The patient experienced recurrent carpopedal spasms after six hours of daily tennis.
- Serum magnesium levels were found to be low (Hypomagnesemia).
- Magnesium supplementation led to the resolution of carpopedal spasms and normalization of serum magnesium.
Conclusions:
- Hypomagnesemia can manifest as carpopedal spasm in individuals undertaking extreme endurance exercise.
- Magnesium repletion is an effective treatment for exercise-induced carpopedal spasm secondary to Hypomagnesemia.
- This case underscores the importance of assessing magnesium status in athletes with unexplained muscle cramps or spasms.
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