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Bilateral stage I chronic maxillary atelectasis: A case report.

Dakshika A Gunaratne¹, Zubair Hasan¹, Peter Floros¹

¹Department of Otolaryngology, Head and Neck Surgery, Westmead Hospital, Sydney, Australia.

International Journal of Surgery Case Reports

|July 25, 2016

Summary

Chronic maxillary atelectasis (CMA) is a rare condition causing sinus volume reduction. This case report details successful endoscopic treatment for a rare bilateral presentation of stage I CMA.

Keywords:

Chronic maxillary atelectasis Endoscopic sinus surgery Maxillary sinus Sinusitis

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Area of Science:

Otolaryngology
Rhinology
Surgical Anatomy

Background:

Chronic maxillary atelectasis (CMA) is a rare condition characterized by progressive maxillary sinus volume reduction and antral wall collapse.
It results from ostiomeatal obstruction and negative intra-sinus pressure gradients.
Existing literature on CMA, including terms like 'silent sinus syndrome,' is inconsistent.

Purpose of the Study:

To report a unique case of bilateral stage I Chronic maxillary atelectasis (CMA).
To highlight the successful endoscopic surgical management of this rare condition.
To contribute to the limited literature on bilateral CMA and its rapid progression.

Main Methods:

A 32-year-old male presented with persistent sinonasal symptoms post-upper respiratory tract infection.
Imaging confirmed bilateral stage I Chronic maxillary atelectasis (CMA).
Treatment involved bilateral endoscopic uncinectomy and maxillary antrostomy.

Main Results:

Successful treatment of bilateral stage I Chronic maxillary atelectasis (CMA) was achieved.
The patient experienced symptomatic relief and halted disease progression.
This represents the first reported case of bilateral stage I CMA with an established inciting event and rapid progression.

Conclusions:

Minimally invasive endoscopic surgery can effectively treat Chronic maxillary atelectasis (CMA).
Endoscopic approaches re-establish sinus aeration, halt progression, and promote sinus remodeling.
Further research is needed to fully understand the etiology and pathogenesis of CMA.