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Related Concept Videos

Long-term Potentiation01:35

Long-term Potentiation

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Long-term potentiation, or LTP, is one of the ways by which synaptic plasticity—changes in the strength of chemical synapses—can occur in the brain. LTP is the process of synaptic strengthening that occurs over time between pre- and postsynaptic neuronal connections. The synaptic strengthening of LTP works in opposition to the synaptic weakening of long-term depression (LTD) and together are the main mechanisms that underlie learning and memory.
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Long-term potentiation, or LTP, is one of the ways by which synaptic plasticity—changes in the strength of chemical synapses—can occur in the brain. LTP is the process of synaptic strengthening that occurs over time between pre and postsynaptic neuronal connections. The synaptic strengthening of LTP works in opposition to the synaptic weakening of long-term depression (LTD) and together are the main mechanisms that underlie learning and memory.
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In humans, more than 80% of the genome gets transcribed. However, only around 2% of the genome codes for proteins. The remaining part produces non-coding RNAs which includes ribosomal RNAs, transfer RNAs, telomerase RNAs, and regulatory RNAs, among other types. A large number of regulatory non-coding RNAs have been classified into two groups depending upon their length – small non-coding RNAs, such as microRNA, which are less than 200 nucleotides in length, and long non-coding RNA...
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Long-term depression, or LTD, is one of the ways by which synaptic plasticity—changes in the strength of chemical synapses—can occur in the brain. LTD is the process of synaptic weakening that occurs over time between pre and postsynaptic neuronal connections. The synaptic weakening of LTD works in opposition to synaptic strengthening by long-term potentiation (LTP) and together are the main mechanisms that underlie learning and memory.
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Long-term depression, or LTD, is one of the ways by which synaptic plasticity—changes in the strength of chemical synapses—can occur in the brain. LTD is the process of synaptic weakening that occurs over time between pre and postsynaptic neuronal connections. The synaptic weakening of LTD works in opposition to synaptic strengthening by long-term potentiation (LTP) and together are the main mechanisms that underlie learning and memory.
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Three main types of RNA are involved in protein synthesis: messenger RNA (mRNA), transfer RNA (tRNA), and ribosomal RNA (rRNA). These RNAs perform diverse functions and can be broadly classified as protein-coding or non-coding RNA. Non-coding RNAs play important roles in regulating gene expression in response to developmental and environmental changes. Non-coding RNAs in prokaryotes can be manipulated to develop more effective antibacterial drugs for human or animal use.
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Updated: Mar 17, 2026

Investigation of Synaptic Tagging/Capture and Cross-capture using Acute Hippocampal Slices from Rodents
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Corrigendum: Dynamic expression of long noncoding RNAs and repeat elements in synaptic plasticity.

Jesper L V Maag1, Debabrata Panja2, Ida Sporild2

  • 1Genomics and Epigenetics Division, Garvan Institute of Medical ResearchSydney, NSW, Australia; Faculty of Medicine, St Vincent's Clinical School, University of New South WalesSydney, NSW, Australia.

Frontiers in Neuroscience
|July 30, 2016
PubMed
Summary
This summary is machine-generated.

This article provides a correction to previously published content. The correction pertains to information on page 351 of volume 9.

Keywords:
LTP (long term potentiation)long noncoding RNA (lncRNA)rat brainrepeat elementsretrotransposonssynaptic plasticity (LTP/LTD)time-series data

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Area of Science:

  • Scientific publishing
  • Article correction
  • Bibliographic data

Context:

  • Correction notice for a published article
  • Volume 9, page 351
  • Reference to PMID: 26483626

Purpose:

  • To amend and correct the scientific record
  • Ensure accuracy in published literature
  • Provide updated information to readers

Summary:

  • A specific correction is issued for content previously appearing on page 351.
  • The correction is linked to article PMID: 26483626.
  • This notice rectifies an error in the scientific publication.

Impact:

  • Ensures the integrity of scientific literature
  • Facilitates accurate citation and referencing
  • Maintains reader trust in published research