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Micronucleus test with methyl methanesulfonate administered by intraperitoneal injection and oral gavage

T Tsuyoshi1, M Takeuchi, H Hirono

  • 1Research Department, Sunstar Inc., Osaka, Japan.

Mutation Research
|August 1, 1989
PubMed

Insights

Methyl methanesulfonate (MMS) induces micronucleated polychromatic erythrocytes (MNPCEs) via both intraperitoneal injection and oral gavage in mice. Route-specific differences in MNPCE induction were observed, but both administration routes are acceptable for the micronucleus test.

Area of Science:

  • Toxicology
  • Genotoxicity Testing

Background:

  • The micronucleus test is a standard genotoxicity assay.
  • Evaluating different routes of administration is crucial for accurate toxicological assessments.

Purpose of the Study:

  • To compare the efficacy of intraperitoneal injection (i.p.) versus oral gavage (p.o.) administration of methyl methanesulfonate (MMS) in the mouse micronucleus test.
  • To determine if route-dependent differences in genotoxic effects exist.

Main Methods:

  • Conducted acute toxicity and pilot micronucleus studies.
  • Performed a final micronucleus test using two mouse strains (MS/Ae and CD-1) with MMS administered via i.p. and p.o. routes at various doses.
  • Analyzed micronucleated polychromatic erythrocytes (MNPCEs) at a 24-h sampling time.

Main Results:

  • MMS induced MNPCEs via both i.p. and p.o. routes in both mouse strains.
  • Higher MNPCEs were observed with i.p. administration at lower doses (40 and 80 mg/kg) in both strains.
  • At 160 mg/kg, oral gavage showed higher MNPCE induction in MS/Ae mice.
  • Route-related differences in mg/kg became insignificant when normalized to LD50.

Conclusions:

  • Both intraperitoneal injection and oral gavage are acceptable routes for MMS administration in the mouse micronucleus test.
  • While dose-dependent route differences exist, they are not absolute and can be influenced by factors like mouse strain and specific dose.
  • The findings support the flexibility in choosing administration routes for this genotoxicity assay.

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