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Does silybin protect against toxicity induced by polymyxin E in rat kidney?
Sherif M Sabry Hassan1,2,3, Magdy F Youakim2, Ayman Abou Elenein Rizk2
1Department of Anatomy, A.T. Still University-Kirksville College of Osteopathic Medicine, Kirksville, Missouri.
Aims:
Although once a common antibiotic, polymyxin E fell out of favor after reports of its nephrotoxicity. However, recent concerns with gram-negative bacteria, which are resistant to multiple antibiotics, have resulted in increased interest in polymyxin E. Silybin is a known antihepatotoxic drug and may have potential for protecting the kidney from polymyxin E. Therefore, the aim of the current study was to evaluate whether silybin affected the damages produced by polymyxin E on the rat kidney.
Methods:
Four groups of rats with 10 rats per group were included in the study: control (no treatment, group I), vehicle (control vehicle treatment, group II), polymyxin E treatment (group III), and polymyxin E and silybin treatment (group IV). Groups II-IV received intravenous treatment twice a day for 7 days. All rats were euthanized after 7 days. Histological, ultrastructural, and morphometric analyses were performed on the rats' kidney tissues.
Results:
Analysis of tissues from group III showed differences from groups I and II, such as glomerular and tubular affection and changes in morphometric measures. Results for group IV were more similar to those of groups I and II than those of group III.
Conclusions:
Our results suggested that administering silybin with polymyxin E alleviated polymyxin E-induced nephrotoxicity in the rat kidney. Future biochemical studies should investigate whether silybin could ameliorate the nephrotoxicity caused by polymyxin E in rats and whether concomitant administration of silybin could be an effective clinical pharmacological strategy to protect against polymyxin E-induced insult in human kidneys.
Insights
Silybin administration with polymyxin E reduced kidney damage in rats. This suggests silybin may protect against polymyxin E-induced nephrotoxicity, offering potential clinical applications.
Area of Science:
- Nephrology
- Pharmacology
- Toxicology
Background:
- Polymyxin E, an antibiotic, has seen renewed interest due to rising multidrug-resistant Gram-negative bacteria.
- Polymyxin E use is limited by its significant nephrotoxicity.
- Silybin, known for its liver-protective effects, is being investigated for potential kidney protection.
Purpose of the Study:
- To investigate the protective effects of silybin against polymyxin E-induced kidney damage in a rat model.
- To evaluate the impact of co-administering silybin and polymyxin E on renal tissue.
Main Methods:
- Four groups of rats (n=10/group) were used: control, vehicle, polymyxin E, and polymyxin E + silybin.
- Intravenous treatments were administered for 7 days.
- Kidney tissues underwent histological, ultrastructural, and morphometric analyses.
Main Results:
- Polymyxin E treatment alone induced significant glomerular and tubular damage and altered morphometric measures.
- Rats receiving both polymyxin E and silybin showed kidney tissue characteristics more similar to control groups.
- Silybin co-administration appeared to mitigate the adverse renal effects of polymyxin E.
Conclusions:
- Silybin demonstrated a protective effect against polymyxin E-induced nephrotoxicity in rats.
- Further research is warranted to explore silybin's biochemical mechanisms and its potential as a clinical strategy to prevent polymyxin E-induced kidney damage in humans.
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