Rig-G is a growth inhibitory factor of lung cancer cells that suppresses STAT3 and NF-κB

Dong Li1, Junjun Sun1, Wenfang Liu2

  • 1Department of Clinical Laboratory, Shanghai Tongji Hospital, Tongji University School of Medicine, 200065 Shanghai, China.

Oncotarget
|September 8, 2016
PubMed

Insights

Retinoic acid-induced G (Rig-G) acts as a tumor suppressor in lung cancer. Rig-G inhibits cancer cell proliferation and tumor growth by impacting key signaling pathways like NF-κB.

Area of Science:

  • Oncology
  • Molecular Biology
  • Gene Expression

Background:

  • The retinoic acid-induced G (Rig-G) gene is expressed in various cancer cells, including lung cancer.
  • Its role as a tumor suppressor in lung cancer remains largely uncharacterized.

Purpose of the Study:

  • To investigate the function of Rig-G in lung cancer.
  • To elucidate the molecular mechanisms underlying Rig-G's potential antitumor effects.

Main Methods:

  • Ectopic expression and knockdown of Rig-G in lung cancer cells.
  • Cell proliferation and colony formation assays.
  • Transcriptome and pathway analyses, including NF-κB signaling investigation.

Main Results:

  • Ectopic Rig-G expression significantly decreased lung cancer cell proliferation and inhibited tumor growth.
  • Rig-G knockdown modestly increased cell proliferation and colony formation.
  • Rig-G impacts growth signaling pathways, notably inhibiting NF-κB activity by suppressing STAT3, downregulating miR21 and miR181b-1, and activating the PTEN/Akt and CYLD/IκB signaling axis.

Conclusions:

  • Rig-G functions as a tumor suppressor in lung cancer.
  • Rig-G exerts its antitumor effects by modulating critical signaling pathways, offering a potential therapeutic strategy for lung cancer.

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