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Updated: Mar 15, 2026

Paradigms of Lower Extremity Electrical Stimulation Training After Spinal Cord Injury
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Lower Extremity Paralysis.

D Luke Glancy1, Pramilla N Subramaniam1, Juan F Rodriguez2

  • 1Section of Cardiology, Department of Medicine, Louisiana State University Health Sciences Center, New Orleans, Louisiana.

The American Journal of Cardiology
|September 17, 2016
PubMed
Summary
This summary is machine-generated.

Severe hypokalemia, a condition of low potassium, can cause significant muscle weakness and paralysis. Prompt potassium repletion effectively reversed these symptoms and electrocardiographic changes within 24 hours.

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Area of Science:

  • Internal Medicine
  • Clinical Biochemistry
  • Cardiology

Background:

  • Hypokalemia can arise from various causes, including gastrointestinal losses.
  • Severe electrolyte disturbances require prompt diagnosis and management.
  • The impact of isolated hypokalemia on neuromuscular and cardiac function is significant.

Observation:

  • A 33-year-old male presented with generalized weakness and flaccid paralysis of lower extremities.
  • Electrocardiographic changes and biochemical evidence of rhabdomyolysis were noted.
  • These symptoms were attributed to severe hypokalemia resulting from diarrhea.

Findings:

  • The patient exhibited profound hypokalemia without other significant electrolyte imbalances.
  • Skeletal and cardiac rhabdomyolysis were mild but biochemically evident.
  • Potassium repletion led to the complete resolution of all clinical and biochemical abnormalities.

Implications:

  • This case highlights the critical role of potassium in maintaining neuromuscular and cardiac function.
  • Early recognition and treatment of hypokalemia are essential to prevent severe complications.
  • Diarrhea is a crucial cause of hypokalemia to consider in patients with paralysis and ECG changes.