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Effects of leptospiral lipopolysaccharide on rabbit platelets

E Isogai1, H Kitagawa, H Isogai

  • 1Dept. of Preventive Dentistry, School of Dentistry, Higashi Nippon Gakuen Univ., Hokkaido, Japan.

Zentralblatt Fur Bakteriologie : International Journal of Medical Microbiology
|July 1, 1989
PubMed

Insights

Leptospiral lipopolysaccharides (LPSs) from three Leptospira serovars induced platelet aggregation and lysis. The LPS from serovar copenhageni showed the highest platelet aggregation and cytotoxicity.

Area of Science:

  • Microbiology
  • Immunology
  • Hematology

Background:

  • Leptospiral lipopolysaccharides (LPSs) are key virulence factors in Leptospira infections.
  • Platelets play a crucial role in hemostasis and immune responses.
  • The interaction between bacterial LPS and platelets is not fully understood.

Purpose of the Study:

  • To investigate the effects of Leptospira interrogans LPS on rabbit platelets.
  • To compare the platelet-aggregating and cytotoxic potential of LPS from different Leptospira serovars.

Main Methods:

  • Leptospiral LPS extracted from three virulent strains (serovar copenhageni, canicola, and hebdomadis).
  • Platelet-rich plasma (PRP) from rabbits used for aggregation and lysis assays.
  • Transmission electron microscopy (TEM) and scanning electron microscopy (SEM) for ultrastructural analysis.
  • Measurement of adenosine triphosphate (ATP) and serotonin release.

Main Results:

  • All tested LPSs induced platelet aggregation and released ATP and serotonin.
  • LPS from Leptospira interrogans serovar copenhageni (I-LPS) exhibited the highest platelet aggregation.
  • TEM and SEM revealed platelet surface changes, including pseudopodia formation, upon I-LPS treatment.
  • I-LPS demonstrated significant cytotoxicity, causing degenerative or lytic changes in 44.5% of platelets within 60 minutes.

Conclusions:

  • Leptospiral LPS can activate and damage platelets.
  • Serovar copenhageni LPS is particularly potent in inducing platelet aggregation and cytotoxicity.
  • These findings suggest a role for platelet activation and damage in leptospirosis pathogenesis.

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