Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Concept Videos

Heart Failure III: Clinical Manifestations01:26

Heart Failure III: Clinical Manifestations

761
Heart failure (HF) manifests primarily as dyspnea, fatigue, and fluid retention, resulting in peripheral and pulmonary edema. Symptoms may vary depending on which ventricle is more affected, left or right.Left-Sided Heart FailureAlso known as left ventricular failure, this condition results from the left ventricle's inability to fill or eject sufficient blood into the systemic circulation. It leads to pulmonary congestion, which occurs when the left ventricle fails to eject blood effectively...
761
Angina I: Introduction01:30

Angina I: Introduction

646
Definition and Symptoms: Angina (angina pectoris) is chest pain or discomfort caused by myocardial ischemia, which occurs when the heart muscle receives insufficient oxygen-rich blood. It typically manifests as pressing, squeezing, or crushing sensations in the chest and may radiate to the shoulders, arms, neck, jaw, or back.Primary Cause: In a healthy state, the coronary arteries can dilate (widen) to increase blood flow and meet the increased oxygen demand during physical activity or...
646
Assessment of the Cardiovascular System III: Palpation01:27

Assessment of the Cardiovascular System III: Palpation

1.4K
Palpation involves feeling the body to evaluate texture, size, consistency, and tenderness for assessing cardiovascular health. The following steps are organized in a head-to-toe order:
Jugular Venous Pressure (JVP) Measurement
Position the patient at a thirty- to forty-five-degree angle or in a semi-fowler's position. Look for the highest point of pulsation in the internal jugular vein and measure the vertical distance to the angle of Loius or sternal angle. A normal JVP is 3-4 cm above...
1.4K
Angina III: Clinical Manifestations and Assessment01:29

Angina III: Clinical Manifestations and Assessment

324
Angina manifests as chest pain, tightness, or squeezing discomfort typically located behind the breastbone. It can radiate to the neck, jaw, shoulders, and inner aspects of the upper arms, most commonly the left arm. Patients may experience shortness of breath, fatigue, profuse sweating, dizziness, indigestion, heartburn, palpitations, anxiety, and vomiting as accompanying symptoms. This pain often lasts a few minutes and is triggered by physical exertion, emotional stress, heavy meals, or cold...
324
Angina IV: Management01:26

Angina IV: Management

373
IntroductionThe management of angina requires a comprehensive approach that includes pharmacological therapies, medical procedures, and lifestyle modifications.Pharmacological TherapiesAntiplatelet agents, such as aspirin, clopidogrel, prasugrel, and ticagrelor, play a pivotal role in preventing thrombus formation in patients with angina. These medications inhibit platelet aggregation and reduce the likelihood of myocardial infarction and other cardiovascular events.Anticoagulants, including...
373
Angina II: Classification01:27

Angina II: Classification

515
Angina, also known as angina pectoris, is a chest pain resulting from diminished blood flow to the heart muscle and is often a symptom of coronary artery disease. Angina presents several variants with distinctive attributes, etiologies, and therapeutic approaches. The main types of angina include stable, unstable, variant (Prinzmetal's), microvascular, intractable, and silent ischemia.Stable angina is caused by atherosclerosis, which leads to the formation of plaques that narrow the coronary...
515

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

Risk calculator of the clinical response to antihistamines in chronic urticaria: Development and internal validation.

PloS one·2024
Same author

Bone involvement in non-congenital syphilis

Biomedica : revista del Instituto Nacional de Salud·2023
Same author

Nasal Provocation Challenges with Aedes aegypti Whole-Body Extract Induces Allergic Rhinitis.

International archives of allergy and immunology·2023
Same author

Prospective analysis of clinical evolution in chronic urticaria: Persistence, remission, recurrence, and pruritus alone.

The World Allergy Organization journal·2022
Same author

[Urticaria associated with COVID-19 in allergic patients. A case series].

Revista alergia Mexico (Tecamachalco, Puebla, Mexico : 1993)·2021
Same author

Impact of the allergen-specific immunotherapy in pediatric patients with asthma treated at a health institution in Colombia

Biomedica : revista del Instituto Nacional de Salud·2021

Related Experiment Video

Updated: Mar 12, 2026

Author Spotlight: Unveiling Prognostic Indicators in Heart Failure - The Role of Phase Angle and Bioelectrical Impedance Analysis
04:05

Author Spotlight: Unveiling Prognostic Indicators in Heart Failure - The Role of Phase Angle and Bioelectrical Impedance Analysis

Published on: June 30, 2023

2.9K

[Angioedema].

Luisa Holguín-Gómez1, Luz Adriana Vásquez-Ochoa, Ricardo Cardona

  • 1Universidad de Antioquia. Medellín, Antioquia, Colombia. luisaholguing@gmail.com.

Revista Alergia Mexico (Tecamachalco, Puebla, Mexico : 1993)
|November 1, 2016
PubMed
Summary

Angioedema is swelling of skin or mucosa, typically resolving within 72 hours. Treatment varies by cause, including antihistamines for histaminergic types and specific therapies for hereditary or ACE inhibitor-induced angioedema.

Keywords:
AngioedemaBradykininCyclooxygenaseHistamineIdiopathic angioedemaNonsteroidal anti-inflammatory drugsUrticaria

More Related Videos

Visualizing Leukocyte Rolling and Adhesion in Angiotensin II-Infused Mice: Techniques and Pitfalls
10:16

Visualizing Leukocyte Rolling and Adhesion in Angiotensin II-Infused Mice: Techniques and Pitfalls

Published on: January 4, 2018

9.2K
Subcutaneous Angiotensin II Infusion using Osmotic Pumps Induces Aortic Aneurysms in Mice
07:21

Subcutaneous Angiotensin II Infusion using Osmotic Pumps Induces Aortic Aneurysms in Mice

Published on: September 28, 2015

39.7K

Related Experiment Videos

Last Updated: Mar 12, 2026

Author Spotlight: Unveiling Prognostic Indicators in Heart Failure - The Role of Phase Angle and Bioelectrical Impedance Analysis
04:05

Author Spotlight: Unveiling Prognostic Indicators in Heart Failure - The Role of Phase Angle and Bioelectrical Impedance Analysis

Published on: June 30, 2023

2.9K
Visualizing Leukocyte Rolling and Adhesion in Angiotensin II-Infused Mice: Techniques and Pitfalls
10:16

Visualizing Leukocyte Rolling and Adhesion in Angiotensin II-Infused Mice: Techniques and Pitfalls

Published on: January 4, 2018

9.2K
Subcutaneous Angiotensin II Infusion using Osmotic Pumps Induces Aortic Aneurysms in Mice
07:21

Subcutaneous Angiotensin II Infusion using Osmotic Pumps Induces Aortic Aneurysms in Mice

Published on: September 28, 2015

39.7K

Area of Science:

  • Immunology
  • Dermatology
  • Allergy

Background:

  • Angioedema involves swelling due to increased vascular permeability.
  • It affects skin and mucosal tissues, including respiratory and gastrointestinal tracts.
  • Episodes are generally self-limiting, resolving within 72 hours.

Purpose of the Study:

  • To define angioedema and outline its various types.
  • To describe the underlying pathophysiology of plasma extravasation.
  • To summarize current treatment strategies based on angioedema etiology.

Main Methods:

  • Review of angioedema pathophysiology and classification.
  • Analysis of treatment modalities for different angioedema subtypes.
  • Correlation of treatment with specific causes, patient age, and symptom severity.

Main Results:

  • Identified distinct angioedema types: histaminergic, hereditary, C1 inhibitor deficiency-related, ACE inhibitor-induced, bradykinin-mediated, and idiopathic.
  • Pathophysiology involves increased permeability of capillaries and venules.
  • Treatment approaches range from trigger avoidance and antihistamines to protein replacement or bradykinin antagonism.

Conclusions:

  • Angioedema management requires accurate diagnosis of the underlying cause.
  • Tailored treatment is essential for effective symptom control and prevention.
  • Understanding the diverse mechanisms of angioedema guides therapeutic decisions.