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Related Experiment Videos

[Aortal defects in systemic lupus erythematosus].

Z S Alekberova, G P Kotel'nikova, M Iu Folomeev

    Terapevticheskii Arkhiv
    |January 1, 1989
    PubMed
    Summary

    This study examines aortal valvular disease in patients with systemic lupus erythematosus (SLE). It explores the causes, differentiating between lupus and bacterial endocarditis, and presents diagnostic criteria.

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    [Behçet's disease: Intracardiac thrombosis (a description of two cases and a review of literature)].

    Terapevticheskii arkhiv·2017

    Area of Science:

    • Cardiology
    • Rheumatology
    • Immunology

    Background:

    • Systemic lupus erythematosus (SLE) can affect cardiac structures.
    • Aortal valvular disease is a potential complication in SLE patients.
    • Distinguishing cardiac involvement in SLE from other conditions like bacterial endocarditis is clinically important.

    Purpose of the Study:

    • To describe aortal valvular disease in five patients with significant SLE.
    • To investigate the potential causes of aortal valvular disease in SLE patients, including the lupus process itself versus superimposed bacterial endocarditis.
    • To outline differential and diagnostic criteria for aortal valvular disease in the context of SLE.

    Main Methods:

    • Electrocardiography (ECG)
    • Phonocardiography (PCG)
    • Ultracardiosonography (echocardiography)

    Main Results:

    • Diagnosis of aortal valvular disease was confirmed using ECG, PCG, and echocardiography in five SLE patients.
    • The study discusses the differential diagnosis between lupus-related valvular disease and bacterial endocarditis.
    • Four patients presented with bilateral sacroiliitis and were negative for HLA-B27.

    Conclusions:

    • Aortal valvular disease can occur in patients with SLE.
    • Understanding the pathogenesis of valvular disease in SLE is crucial for appropriate management.
    • Diagnostic tools like echocardiography are essential for identifying and characterizing cardiac involvement in SLE.

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