Glucocorticoids may compromise the effect of gefitinib in non-small cell lung cancer

Hsian-Yu Wang1,2, Yu-Ling Chang3, Chun-Chun Cheng4

  • 1Institute of Biotechnology and Pharmaceutical Research, National Health Research Institutes, Miaoli County, Taiwan.

Oncotarget
|November 12, 2016
PubMed

Insights

Glucocorticoids (GCs) may reduce the effectiveness of epidermal growth factor receptor (EGFR) tyrosine kinase inhibitors (TKIs) in non-small cell lung cancer (NSCLC). This study found that combining GCs with EGFR-TKIs increased disease progression risk in NSCLC patients.

Area of Science:

  • Oncology
  • Pharmacology
  • Cancer Research

Background:

  • Epidermal growth factor receptor (EGFR)-targeting tyrosine kinase inhibitors (TKIs) are effective for non-small cell lung cancer (NSCLC) with sensitive mutations.
  • Glucocorticoids (GCs) are commonly used in cancer patients but may interact with therapies.

Purpose of the Study:

  • To investigate the potential detrimental effects of concomitant Glucocorticoids (GCs) use on EGFR-tyrosine kinase inhibitor (TKI) therapy in non-small cell lung cancer (NSCLC).

Main Methods:

  • Utilized cell cultures and xenograft models to assess GCs' impact on EGFR-TKI efficacy.
  • Conducted a retrospective population study using Taiwan's national health insurance data.

Main Results:

  • Glucocorticoids (GCs) were found to compromise the anti-cancer efficacy of tyrosine kinase inhibitors (TKIs) in NSCLC cell lines and xenografts.
  • Co-medication with GCs was associated with a significantly higher risk of disease progression in NSCLC patients receiving EGFR-TKIs.

Conclusions:

  • Concomitant use of Glucocorticoids (GCs) may negatively impact the effectiveness of EGFR-tyrosine kinase inhibitor (TKI) treatment in non-small cell lung cancer (NSCLC).
  • Further research is warranted to understand the mechanisms and clinical implications of this drug interaction.

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