[Functional restructuring of the bush-like receptor during the suppression of anaerobic glycolysis]

The bush-like receptors of frog's bladder have been investigated simultaneously by vital microscopic, and spectrophotometric studies and registration of biopotentials. It is demonstrated that electrogenic function of receptors is suppressed by monoiodacetate and anoxia more rapidly than without monoiodacetate. At the same time reducing of methylene blue into receptors is accelerated. This demonstrates a damage of power metabolism of sensory terminals. It is suggested that the changes result from blockade of anaerobic glycolysis being an alternative way for power maintenance of receptor function under anoxia.