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Production of interleukin-1-alpha and -beta by human peripheral polymorphonuclear neutrophils

K Goh1, S Furusawa, Y Kawa

  • 1Department of Dermatology, Teikyo University School of Medicine, Tokyo, Japan.

International Archives of Allergy and Applied Immunology
|January 1, 1989
PubMed

Insights

Human neutrophils stimulated with lipopolysaccharide (LPS) produce both interleukin-1 alpha and beta (IL-1α/β). Extracellular IL-1β is released early, while IL-1α is released later, with intracellular IL-1α detected during intermediate periods.

Area of Science:

  • Immunology
  • Cell Biology

Background:

  • Interleukin-1 (IL-1) is a key cytokine in immune responses.
  • Polymorphonuclear neutrophils (PMN) are crucial immune cells involved in inflammation.
  • Understanding IL-1 production mechanisms by PMN is vital for inflammatory disease research.

Purpose of the Study:

  • To investigate the mechanism of interleukin-1 (IL-1) production by human peripheral polymorphonuclear neutrophils (PMN).
  • To characterize the temporal release and localization of IL-1 alpha and IL-1 beta isoforms.

Main Methods:

  • PMN were stimulated with lipopolysaccharide (LPS).
  • Extracellular IL-1 was obtained from supernatants, and intracellular IL-1 from lysate.
  • IL-1 activity was quantified using the C3H/HeJ thymocyte co-mitogenic assay.
  • IL-1 isoforms were characterized by molecular weight, pI, and neutralization with specific antibodies.

Main Results:

  • LPS-stimulated PMN produced IL-1 with molecular weight of 15-20 kDa and pI values of 5.0 and >8.5.
  • The produced IL-1 activity was neutralized by anti-IL-1 antibodies and lacked IL-2 activity.
  • Extracellular IL-1 beta was predominantly detected in early incubation periods, while IL-1 alpha appeared later.
  • Intracellular IL-1 alpha was mainly detected during intermediate incubation periods, whereas IL-1 beta was not detected intracellularly.

Conclusions:

  • Human PMN stimulated with LPS produce both IL-1 alpha and IL-1 beta.
  • PMN release IL-1 beta extracellularly before releasing IL-1 alpha.
  • These findings elucidate the distinct temporal dynamics of IL-1 isoform production and release by neutrophils.

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