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Mouse epidermal growth factor concentrations are altered by gonadectomy and treatments with estradiol and

T Tuomela1, L Viinikka, J Perheentupa

  • 1Children's Hospital, University of Helsinki, Finland.

Life Sciences
|January 1, 1989
PubMed

Insights

Gonadectomy alters epidermal growth factor (EGF) levels in mice. Estradiol and progesterone treatments modify these EGF concentrations in salivary glands, kidneys, plasma, and urine.

Area of Science:

  • Endocrinology
  • Molecular Biology
  • Physiology

Background:

  • Epidermal Growth Factor (EGF) plays a crucial role in cellular processes.
  • Gonadal hormones are known to influence various physiological functions.
  • The interaction between gonadal steroids and EGF in adult mice requires further elucidation.

Purpose of the Study:

  • To compare epidermal growth factor (EGF) concentrations in gonadectomized male and female mice.
  • To investigate the effects of post-gonadectomy treatments with estradiol and progesterone on EGF levels.

Main Methods:

  • Adult male and female mice underwent gonadectomy.
  • Submandibular salivary gland (SMG), kidney, plasma, and urine samples were collected.
  • EGF concentrations were measured using established biochemical assays.
  • Mice received post-gonadectomy treatments with estradiol, progesterone, or a combination.

Main Results:

  • Gonadectomized females exhibited significantly higher EGF concentrations in the SMG compared to males (7-fold).
  • Males showed higher EGF levels in kidneys (1.3-fold) and plasma, while females had higher urinary EGF.
  • Estradiol treatment decreased SMG EGF and increased urinary and kidney EGF.
  • Progesterone affected male kidney EGF, and combined treatments modulated EGF in male urine and SMG.

Conclusions:

  • Gonadectomy significantly impacts EGF distribution across different tissues and biofluids in mice.
  • Estradiol and progesterone differentially modulate EGF concentrations, suggesting a complex hormonal regulation.
  • These findings highlight the influence of sex hormones on EGF homeostasis and have implications for understanding tissue-specific responses.

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