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Sequencing Small Non-coding RNA from Formalin-fixed Tissues and Serum-derived Exosomes from Castration-resistant Prostate Cancer Patients
Published on: November 19, 2019
Potential therapeutic effect of epigenetic therapy on treatment-induced neuroendocrine prostate cancer
Xiang Xu1,2, Yu-Hua Huang3, Yan-Jing Li2
1School of Life Sciences, Anhui University, Hefei, China.
Abstract:
Although adenocarcinomas of the prostate are relatively indolent, some patients with advanced adenocarcinomas show recurrence of treatment-induced neuroendocrine prostate cancer, which is highly aggressive and lethal. Detailed biological features of treatment-induced neuroendocrine prostate cancer have not been characterized owing to limited biopsies/resections and the lack of a cellular model. In this study, we used a unique cellular model (LNCaP/NE1.8) to investigate the potential role of cancer stem cells in treatment-induced neuroendocrine prostate cancer with acquired resistance to hormonal therapy and chemotherapy. We also studied the role of cancer stem cells in enhancing invasion in treatment-induced neuroendocrine prostate cancer cells that recurred after long-term androgen-ablation treatment. Using an in vitro system mimicking clinical androgen-ablation, our results showed that the neuroendocrine-like subclone NE1.8 cells were enriched with cancer stem cells. Compared to parental prostate adenocarcinoma LNCaP cells, NE1.8 cells are more resistant to androgen deprivation therapy and chemotherapeutic agents and show increased cancer cell invasiveness. Results from this study also suggest a potential epigenetic therapeutic strategy using suberoylanilide hydroxamic acid, a histone deacetylase inhibitor, as a chemotherapeutic agent for therapy-resistant treatment-induced neuroendocrine prostate cancer cells to minimize the risk of prostate cancer recurrence and metastasis.
Insights
Treatment-induced neuroendocrine prostate cancer stem cells drive aggressive recurrence and invasion. Epigenetic therapy with suberoylanilide hydroxamic acid may offer a new strategy against therapy-resistant prostate cancer.
Area of Science:
- Oncology
- Cancer Biology
- Molecular Urology
Background:
- Advanced prostate adenocarcinomas can recur as aggressive neuroendocrine prostate cancer.
- Limited biopsies and lack of cellular models hinder understanding of this lethal subtype.
- Cancer stem cells (CSCs) are implicated in treatment resistance and metastasis.
Purpose of the Study:
- Investigate the role of CSCs in treatment-induced neuroendocrine prostate cancer (NEPC).
- Characterize CSC enrichment and function in NEPC with acquired resistance.
- Explore potential therapeutic strategies targeting CSCs in recurrent NEPC.
Main Methods:
- Utilized a unique LNCaP/NE1.8 cellular model mimicking androgen-ablation therapy.
- Enriched neuroendocrine-like subclone NE1.8 cells for CSC analysis.
- Assessed chemoresistance, hormonal therapy resistance, and invasiveness of NE1.8 cells compared to parental LNCaP cells.
Main Results:
- The NE1.8 subclone showed enrichment of CSCs compared to parental cells.
- NE1.8 cells exhibited increased resistance to androgen deprivation and chemotherapy.
- NE1.8 cells demonstrated enhanced invasiveness, suggesting a role in metastasis.
Conclusions:
- CSCs play a significant role in the aggressive behavior and recurrence of treatment-induced NEPC.
- Suberoylanilide hydroxamic acid (SAHA), a histone deacetylase inhibitor, shows potential as a therapeutic agent for therapy-resistant NEPC.
- Targeting CSCs epigenetically may reduce the risk of prostate cancer recurrence and metastasis.
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