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Visual Agnosia

Visual Agnosia

Visual agnosia is a condition characterized by the inability to recognize visually presented objects despite having normal vision. For instance, a person with visual agnosia can describe the shape and color of an object but cannot identify or name it. This impairment does not affect their visual field, acuity, color vision, brightness discrimination, language, or memory. An example of this condition in a social setting is someone at a dinner party asking for "that silver thing with a round...

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Idiopathic Acquired Temporal Wedge Visual Field Defects.

Michael J Gilhooley¹, Clare L Fraser², Sui Wong¹

¹Moorfields Eye Hospital , London, United Kingdom.

Neuro-Ophthalmology (Aeolus Press)

|December 9, 2016

Summary

This summary is machine-generated.

This study describes 13 unique cases of acquired, stationary temporal wedge scotomas, a novel finding in ophthalmology. The cause is unknown, but damage to the optic disc

Keywords:

Temporal defects field scotomas wedge

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Area of Science:

Ophthalmology
Neuro-ophthalmology

Background:

Stationary "wedge" visual field defects have been previously associated with congenital nasal hypoplasia of the optic disc or vitreoretinal surgery complications.
Spontaneous occurrence of such defects is not documented in existing literature.

Purpose of the Study:

To report 13 unusual cases of acquired, stationary, temporal sectoral scotomas.
To investigate the clinical characteristics and potential mechanisms of these novel visual field defects.

Main Methods:

Descriptive clinical case series of 13 patients with sub-acute monocular temporal visual field loss.
Investigations included Goldmann perimetry, automated Humphrey visual fields, optical coherence tomography, orbital ultrasound, and electroretinography.
Serial perimetry follow-up for a mean of 3.9 years.

Main Results:

All patients presented with stable, wedge-shaped temporal visual field defects originating near the blind spot.
No evidence of optic disc drusen, glaucoma, disc hypoplasia, or focal retinitis was found.
One patient developed a similar defect in the contralateral eye after 5 years.

Conclusions:

This study presents 13 novel cases of acquired, stationary temporal wedge scotomas.
The etiology remains uncertain, but damage to the nasal rim of the optic disc is hypothesized as a potential mechanism.
Further research is needed to elucidate the underlying cause of these unique visual field defects.