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Cyclosporin-induced embryotoxicity in mice

A Fein1, M Vechoropoulos, L Nebel

  • 1Department of Embryology and Teratology, Tel-Aviv University Medical School, Tel-Hashomer, Israel.

Biology of the Neonate
|January 1, 1989
PubMed

Insights

Cyclosporin (CS) administration during pregnancy in mice did not increase maternal death but caused significant embryotoxicity. The drug reduced viable embryos and increased resorptions, indicating a clear adverse effect on fetal development.

Area of Science:

  • Toxicology
  • Developmental Biology
  • Immunology

Background:

  • Cyclosporin (CS) is an immunosuppressive drug with potential effects on pregnancy.
  • Understanding CS's impact on embryonic development is crucial for risk assessment.

Purpose of the Study:

  • To investigate the embryotoxic effects of cyclosporin (CS) in pregnant ICR mice.
  • To evaluate the impact of CS on maternal organs and embryonic development at various gestational stages.

Main Methods:

  • Pregnant ICR mice received daily injections of 30 mg/kg cyclosporin (CS) on gestational days 6-8 or 10-12.
  • Control groups received saline injections.
  • Mice were sacrificed at different time points (days 12, 15, 17, 19) for analysis.
  • Embryos, placentae, and maternal organs were examined macroscopically and microscopically.

Main Results:

  • Cyclosporin (CS) administration did not increase maternal mortality.
  • Histological examination revealed transient pathological alterations in maternal thymus, liver, kidney, and spleen.
  • CS significantly reduced the number of viable embryos and increased embryo resorptions.
  • Microscopic examination indicated no adverse effects on embryonic organogenesis.

Conclusions:

  • Cyclosporin (CS) at 30 mg/kg exhibits clear embryotoxic effects in ICR mice.
  • The drug impacts pregnancy outcomes by reducing viable fetuses and increasing resorptions.
  • While maternal organs showed transient changes, the primary concern is the drug's adverse effect on embryonic development.

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