Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Experiment Videos

Hyperproduction glaucoma.

A Neetens, M C Rubbens

    Transactions of the Ophthalmological Societies of the United Kingdom
    |September 1, 1977
    PubMed
    Summary
    This summary is machine-generated.

    Hyperproduction glaucoma, marked by high intraocular pressure (IOP) and normal outflow facility, stems from excessive aqueous humor production. Treatment involves medications to reduce aqueous inflow, with glucocorticoids for inflammatory cases.

    Related Concept Videos

    You might also read

    Related Articles

    Articles linked to this work by shared authors, journal, and citation graph.

    Sort by
    Same author

    Fractures of the Skull: Ophthalmological Significance.

    Bulletin of the New York Academy of Medicine·2009
    Same author

    [Critical (foveal) flicker fusion frequency (CFF) is helpful in differential diagnosis of organic lesions from orthoptic amblyopias].

    Klinische Monatsblatter fur Augenheilkunde·1998
    Same author

    Human retinal pigment epithelial cells from different donors continuously produce a vascular endothelial cell-stimulating factor into serum-free medium.

    Journal of cell science·1993
    Same author

    [Optic neuritis in non-Hodgkin's lymphoma].

    Klinische Monatsblatter fur Augenheilkunde·1992
    Same author

    Recent pathogenic concepts of primary open angle glaucoma and soft glaucoma (POAG).

    Bulletin de la Societe belge d'ophtalmologie·1992
    Same author

    Foveal flicker-fusion frequencies: a simple, new apparatus (4F).

    Graefe's archive for clinical and experimental ophthalmology = Albrecht von Graefes Archiv fur klinische und experimentelle Ophthalmologie·1992
    Same journal

    Brain stem and cerebellar deficits in eye movement control.

    Transactions of the ophthalmological societies of the United Kingdom·1986
    Same journal

    Strategies in Doppler ultrasound.

    Transactions of the ophthalmological societies of the United Kingdom·1986
    Same journal

    Congenital glaucoma--diagnosis and management.

    Transactions of the ophthalmological societies of the United Kingdom·1986
    Same journal

    Prostaglandins and other eicosanoids: their ocular transport, pharmacokinetics, and therapeutic effects.

    Transactions of the ophthalmological societies of the United Kingdom·1986
    Same journal

    Development of the ciliary body: a brief review.

    Transactions of the ophthalmological societies of the United Kingdom·1986
    Same journal

    Computer automated visual acuity testing for visual screening.

    Transactions of the ophthalmological societies of the United Kingdom·1986
    See all related articles

    Area of Science:

    • Ophthalmology
    • Glaucoma Research
    • Ocular Physiology

    Background:

    • Hyperproduction glaucoma is a clinical condition defined by elevated intraocular pressure (IOP) and normal C:1 (outflow facility).
    • This condition can manifest as spontaneous open-angle glaucoma or be induced by pharmacological agents.
    • It may also present as inflammatory ocular hypertension, involving a breakdown of the blood-aqueous barrier.

    Purpose of the Study:

    • To elucidate the pathogenesis of hyperproduction glaucoma.
    • To outline the primary treatment strategies for this specific glaucoma subtype.
    • To differentiate between spontaneous and induced forms, including inflammatory presentations.

    Main Methods:

    • Tonographic assessment to determine IOP and outflow facility.

    Related Experiment Videos

  • Clinical observation for spontaneous or pharmacologically induced glaucoma.
  • Evaluation of blood-aqueous barrier integrity in inflammatory cases.
  • Main Results:

    • Hyperproduction glaucoma is characterized by high IOP with normal C:1.
    • The pathogenesis is linked to increased aqueous humor hyperproduction.
    • Increased capillary hydrostatic pressure in the ciliary body is the primary driver.

    Conclusions:

    • Effective treatment involves medications like epinephrine and acetazolamide to inhibit aqueous humor inflow.
    • Glucocorticoids are indicated for inflammatory hyperproduction glaucoma.
    • Understanding the role of ciliary body capillary pressure is key to managing this condition.