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An Integrated Platform for Genome-wide Mapping of Chromatin States Using High-throughput ChIP-sequencing in Tumor Tissues
Published on: April 5, 2018
Recent advances in understanding/assessing toxicity to the epigenome
1Forensic and National Security Sciences Institute, Center for Science & Technology, Syracuse University, Syracuse, NY, USA.
Abstract:
The ability of non-genotoxic agents to induce cancer has been documented and clearly requires a reassessment of testing for environmental and human safety. Drug safety testing has historically relied on test batteries designed to detect DNA damage leading to mutation and cancer. The standard genetic toxicology testing battery has been a reliable tool set to identify small molecules/chemicals as hazards that could lead to genetic changes in organisms and induction of cancer. While pharmaceutical companies and regulatory agencies have extensively used the standard battery, it is not suitable for compounds that may induce epigenetic changes. Additionally, many pharmaceutical companies have changed their product portfolios to include peptides and/or other biological molecules, which are not expected to be genotoxic in their own right. If we are to best use our growing knowledge regarding chemicals and biomolecules that induce heritable changes via epigenetic mechanisms, then we must ask what changes may be needed in our testing paradigm to predict long-term downstream effects through epigenetic mechanisms.
Insights
Non-genotoxic agents can cause cancer, necessitating a reevaluation of safety testing. Current genetic toxicology tests miss epigenetic mechanisms, requiring updated paradigms for comprehensive risk assessment.
Area of Science:
- Toxicology
- Genetics
- Epigenetics
- Drug Safety Evaluation
Background:
- Non-genotoxic agents are increasingly recognized for their potential to induce cancer, challenging traditional safety assessment models.
- Current drug safety testing predominantly relies on genetic toxicology assays focused on DNA damage and mutations.
- The evolving pharmaceutical landscape includes biologics and peptides, which may not be adequately assessed by standard genotoxicity tests.
Purpose of the Study:
- To highlight the limitations of existing genetic toxicology testing batteries in detecting non-genotoxic carcinogens.
- To emphasize the need for reassessing safety testing strategies in light of epigenetic mechanisms of carcinogenesis.
- To advocate for the development of new testing paradigms that incorporate epigenetic endpoints for comprehensive risk assessment.
Main Methods:
- Review of existing literature on non-genotoxic carcinogens and epigenetic mechanisms.
- Analysis of the suitability of current genetic toxicology testing batteries for novel therapeutic modalities.
- Discussion of the implications of epigenetic modifications in chemical and biomolecule safety evaluation.
Main Results:
- Documented evidence shows non-genotoxic agents can induce cancer, indicating a gap in current safety testing.
- Standard genetic toxicology tests are insufficient for identifying compounds that exert effects through epigenetic pathways.
- The shift towards biologics and peptides necessitates a broader approach beyond genotoxicity.
Conclusions:
- A fundamental reassessment of environmental and human safety testing is required.
- Existing testing paradigms must be updated to include the evaluation of epigenetic mechanisms.
- Future safety testing must evolve to predict long-term downstream effects mediated by epigenetic changes for both chemicals and biomolecules.
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