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Capsid protein VP4 of poliovirus is N-myristoylated

A V Paul1, A Schultz, S E Pincus

  • 1Department of Microbiology, School of Medicine, State University of New York at Stony Brook 11794.

Insights

Poliovirus capsid protein VP4 is N-myristoylated, with myristic acid attaching to its N-terminal glycine. This lipid modification of poliovirus VP4 and its precursors may play a role in viral proliferation.

Area of Science:

  • Virology
  • Molecular Biology
  • Biochemistry

Background:

  • Poliovirus is an RNA virus that causes poliomyelitis.
  • Viral capsid proteins play crucial roles in viral structure and replication.
  • Post-translational modifications can significantly impact protein function.

Purpose of the Study:

  • To investigate the nature of the N-terminal modification of poliovirus VP4.
  • To determine if the modification involves lipid acylation.
  • To explore the potential role of this modification in poliovirus proliferation.

Main Methods:

  • In vivo labeling of poliovirus with [3H]myristic acid.
  • Analysis of radiolabeled capsid polypeptides by copurification.
  • Characterization of the N-terminal modification of VP4.

Main Results:

  • [3H]myristic acid residues were found to copurify with poliovirus VP4.
  • Evidence confirmed N-myristoylation of the N-terminal glycine residue of VP4.
  • The N-terminal blocking group, identified as myristic acid, is present on VP4 and its precursors (VP0, P1).

Conclusions:

  • Poliovirus VP4 undergoes N-myristoylation at its N-terminal glycine.
  • This lipid modification is also present on VP4 precursor polypeptides.
  • The functional significance of VP4 N-myristoylation in poliovirus proliferation warrants further investigation.

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