Abelson virus as a probe for prothymocytes

Same author

RIPK1- and RIPK3-induced cell death mode is determined by target availability.

Cell death and differentiation·2014

Same author

Necroptosis induced by RIPK3 requires MLKL but not Drp1.

Cell death & disease·2014

Same author

TNF can activate RIPK3 and cause programmed necrosis in the absence of RIPK1.

Cell death & disease·2013

Same author

Hand knitting, frame knitting and rotary frame knitting in the 17th, 18th and 19th centuries. a question of identification.

Textile history·2012

Same author

In mouse embryonic fibroblasts, neither caspase-8 nor cellular FLICE-inhibitory protein (FLIP) is necessary for TNF to activate NF-κB, but caspase-8 is required for TNF to cause cell death, and induction of FLIP by NF-κB is required to prevent it.

Cell death and differentiation·2011

Same author

Accommodating haploinsufficient tumor suppressor genes in Knudson's model.

Oncogene·2000

Same journal

Emapalumab plus conventional therapy with or without ruxolitinib for pediatric hemophagocytic lymphohistiocytosis: a single center retrospective study.

Immunologic research·2026

Same journal

Immunological spectrum in patients with thymoma: beyond good syndrome.

Immunologic research·2026

Same journal

Microbiome immune crosstalk in Sjögren's syndrome: mechanistic insights and translational perspectives.

Immunologic research·2026

Same journal

Immune checkpoint inhibitor-induced myasthenia gravis and myocarditis: a fatal immune-related adverse event.

Immunologic research·2026

Same journal

TRIM28 and TRIM32: multifaceted regulators of innate immunity and antiviral defence.

Immunologic research·2026

Same journal

Decoding PANoptosis: Crosstalk of cell death pathways in immunity and inflammation.

Immunologic research·2026

Related Experiment Videos

Abelson virus as a probe for prothymocytes

Related Concept Videos

RIPK1- and RIPK3-induced cell death mode is determined by target availability.

Necroptosis induced by RIPK3 requires MLKL but not Drp1.

TNF can activate RIPK3 and cause programmed necrosis in the absence of RIPK1.

Hand knitting, frame knitting and rotary frame knitting in the 17th, 18th and 19th centuries. a question of identification.

In mouse embryonic fibroblasts, neither caspase-8 nor cellular FLICE-inhibitory protein (FLIP) is necessary for TNF to activate NF-κB, but caspase-8 is required for TNF to cause cell death, and induction of FLIP by NF-κB is required to prevent it.

Accommodating haploinsufficient tumor suppressor genes in Knudson's model.

Emapalumab plus conventional therapy with or without ruxolitinib for pediatric hemophagocytic lymphohistiocytosis: a single center retrospective study.

Immunological spectrum in patients with thymoma: beyond good syndrome.

Microbiome immune crosstalk in Sjögren's syndrome: mechanistic insights and translational perspectives.

Immune checkpoint inhibitor-induced myasthenia gravis and myocarditis: a fatal immune-related adverse event.

TRIM28 and TRIM32: multifaceted regulators of innate immunity and antiviral defence.

Decoding PANoptosis: Crosstalk of cell death pathways in immunity and inflammation.

Related Experiment Videos

Abelson virus as a probe for prothymocytes

Related Concept Videos

Related Articles

RIPK1- and RIPK3-induced cell death mode is determined by target availability.

Necroptosis induced by RIPK3 requires MLKL but not Drp1.

TNF can activate RIPK3 and cause programmed necrosis in the absence of RIPK1.

Hand knitting, frame knitting and rotary frame knitting in the 17th, 18th and 19th centuries. a question of identification.

In mouse embryonic fibroblasts, neither caspase-8 nor cellular FLICE-inhibitory protein (FLIP) is necessary for TNF to activate NF-κB, but caspase-8 is required for TNF to cause cell death, and induction of FLIP by NF-κB is required to prevent it.

Accommodating haploinsufficient tumor suppressor genes in Knudson's model.

Emapalumab plus conventional therapy with or without ruxolitinib for pediatric hemophagocytic lymphohistiocytosis: a single center retrospective study.

Immunological spectrum in patients with thymoma: beyond good syndrome.

Microbiome immune crosstalk in Sjögren's syndrome: mechanistic insights and translational perspectives.

Immune checkpoint inhibitor-induced myasthenia gravis and myocarditis: a fatal immune-related adverse event.

TRIM28 and TRIM32: multifaceted regulators of innate immunity and antiviral defence.

Decoding PANoptosis: Crosstalk of cell death pathways in immunity and inflammation.