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Related Concept Videos

Heart Failure III: Clinical Manifestations01:26

Heart Failure III: Clinical Manifestations

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Heart failure (HF) manifests primarily as dyspnea, fatigue, and fluid retention, resulting in peripheral and pulmonary edema. Symptoms may vary depending on which ventricle is more affected, left or right.Left-Sided Heart FailureAlso known as left ventricular failure, this condition results from the left ventricle's inability to fill or eject sufficient blood into the systemic circulation. It leads to pulmonary congestion, which occurs when the left ventricle fails to eject blood effectively...
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Angina I: Introduction01:30

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Definition and Symptoms: Angina (angina pectoris) is chest pain or discomfort caused by myocardial ischemia, which occurs when the heart muscle receives insufficient oxygen-rich blood. It typically manifests as pressing, squeezing, or crushing sensations in the chest and may radiate to the shoulders, arms, neck, jaw, or back.Primary Cause: In a healthy state, the coronary arteries can dilate (widen) to increase blood flow and meet the increased oxygen demand during physical activity or...
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Assessment of the Cardiovascular System III: Palpation01:27

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Palpation involves feeling the body to evaluate texture, size, consistency, and tenderness for assessing cardiovascular health. The following steps are organized in a head-to-toe order:
Jugular Venous Pressure (JVP) Measurement
Position the patient at a thirty- to forty-five-degree angle or in a semi-fowler's position. Look for the highest point of pulsation in the internal jugular vein and measure the vertical distance to the angle of Loius or sternal angle. A normal JVP is 3-4 cm above...
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Angina III: Clinical Manifestations and Assessment01:29

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Angina manifests as chest pain, tightness, or squeezing discomfort typically located behind the breastbone. It can radiate to the neck, jaw, shoulders, and inner aspects of the upper arms, most commonly the left arm. Patients may experience shortness of breath, fatigue, profuse sweating, dizziness, indigestion, heartburn, palpitations, anxiety, and vomiting as accompanying symptoms. This pain often lasts a few minutes and is triggered by physical exertion, emotional stress, heavy meals, or cold...
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Angina IV: Management01:26

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IntroductionThe management of angina requires a comprehensive approach that includes pharmacological therapies, medical procedures, and lifestyle modifications.Pharmacological TherapiesAntiplatelet agents, such as aspirin, clopidogrel, prasugrel, and ticagrelor, play a pivotal role in preventing thrombus formation in patients with angina. These medications inhibit platelet aggregation and reduce the likelihood of myocardial infarction and other cardiovascular events.Anticoagulants, including...
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Angina II: Classification01:27

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Angina, also known as angina pectoris, is a chest pain resulting from diminished blood flow to the heart muscle and is often a symptom of coronary artery disease. Angina presents several variants with distinctive attributes, etiologies, and therapeutic approaches. The main types of angina include stable, unstable, variant (Prinzmetal's), microvascular, intractable, and silent ischemia.Stable angina is caused by atherosclerosis, which leads to the formation of plaques that narrow the coronary...
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Author Spotlight: Unveiling Prognostic Indicators in Heart Failure - The Role of Phase Angle and Bioelectrical Impedance Analysis
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Angioedema.

Daniel LoVerde1, Daniel Clark Files, Guha Krishnaswamy

  • 11Division of Pulmonary, Critical Care, Allergy and Immunology, Department of Medicine, Wake Forest Baptist Medical Center, Winston-Salem, NC.2Division of Allergy and Clinical Immunology, Department of Medicine, W.G. (Bill) Hefner VA Medical Center, Salisbury, NC.

Critical Care Medicine
|March 15, 2017
PubMed
Summary
This summary is machine-generated.

Angioedema is a serious condition with various subtypes. This review covers genetics, pathophysiology, and treatments, emphasizing new medications for acute angioedema management.

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Area of Science:

  • Critical care medicine
  • Pharmacology
  • Genetics

Background:

  • Angioedema is a potentially life-threatening condition requiring critical care.
  • Recent advances have improved understanding of angioedema pathophysiology and treatment outcomes.
  • Novel pharmacologic agents are now available for managing angioedema syndromes.

Purpose of the Study:

  • To review the genetics, pathophysiology, classification, and treatment of angioedema syndromes.
  • To emphasize novel pharmacologic agents for acute angioedema treatment.
  • To provide a clinically focused overview for critical care providers.

Main Methods:

  • A MEDLINE search was performed using relevant MeSH terms.
  • Publications on angioedema diagnosis, management, and genetics were selected.
  • Data from retrieved publications were synthesized for review.

Main Results:

  • The current state of diagnosis and management for various angioedema syndromes was described.
  • Information on underlying genetics and pathophysiology was synthesized.
  • Novel pharmacologic interventions were highlighted.

Conclusions:

  • Angioedema encompasses multiple subtypes with distinct pathophysiologies.
  • An evidence-based approach to diagnosis and management is presented.
  • Airway management and targeted pharmacologic interventions are crucial for angioedema treatment.