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Exercise-Associated Hyponatremia: 2017 Update.

Tamara Hew-Butler1, Valentina Loi2, Antonello Pani2

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|March 21, 2017
PubMed
Summary
This summary is machine-generated.

Exercise-associated hyponatremia (EAH) is often caused by drinking too much fluid and arginine vasopressin release. Recent research expands understanding of EAH causes, effects like rhabdomyolysis, and prevention strategies for athletes.

Keywords:
exercisehyponatremiapathogenesisrhabdomyolysiswater

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Area of Science:

  • Sports Medicine
  • Exercise Physiology
  • Endocrinology

Background:

  • Exercise-associated hyponatremia (EAH) was first identified in endurance athletes.
  • Overhydration and arginine vasopressin (AVP) release are primary causes.
  • EAH is increasingly recognized across diverse athletic events and linked to rhabdomyolysis.

Purpose of the Study:

  • To review recent scientific literature on EAH.
  • To summarize novel findings in EAH epidemiology, pathophysiology, treatment, and prevention.

Main Methods:

  • Literature review of recent studies on EAH.
  • Synthesis of current evidence on EAH etiology and clinical manifestations.

Main Results:

  • Overdrinking beyond thirst and non-osmotic AVP release remain key factors.
  • EAH is observed in a wider range of sports.
  • Volume and sodium depletion may contribute in some athletes.
  • EAH is associated with rhabdomyolysis development.

Conclusions:

  • Understanding EAH requires considering fluid balance, AVP, and electrolyte status.
  • Prevention and treatment strategies should be tailored to individual athlete needs.
  • Further research is needed to fully elucidate EAH mechanisms and optimize management.