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Regulation of feeding behaviour in monosodium glutamate (MSG) treated mice

V Ma1, A Ma, D P Cameron

  • 1Department of Diabetes and Endocrinology, Princess Alexandra Hospital, Woolloongabba, Queensland, Australia.

Life Sciences
|January 1, 1988
PubMed

Insights

Neonatal monosodium glutamate (MSG) treatment in mice affects food intake patterns. However, opioid kappa receptor mechanisms regulating appetite remain functional in MSG-treated mice.

Area of Science:

  • Neuroscience
  • Endocrinology
  • Behavioral Science

Background:

  • Monosodium glutamate (MSG) is a common food additive.
  • Neonatal exposure to MSG can alter neurodevelopment and physiological functions.
  • Understanding the long-term effects of MSG on food intake regulation is crucial.

Purpose of the Study:

  • To investigate the impact of neonatal MSG treatment on the regulation of food intake in mice.
  • To assess the role of opioid kappa receptors in the feeding behavior of MSG-treated mice.

Main Methods:

  • Mice were treated neonatally with MSG.
  • Food intake patterns (diurnal and nocturnal) were monitored.
  • Responses to restricted food availability were evaluated.
  • The effect of a kappa opioid receptor agonist (ketocyclazocine) on food intake was assessed.

Main Results:

  • MSG-treated mice exhibited a diurnal feeding pattern with greater nocturnal intake.
  • While MSG mice increased food intake during food restriction, it was less efficient than controls.
  • Ketocyclazocine administration enhanced food intake in MSG-treated mice.

Conclusions:

  • Neonatal MSG treatment alters diurnal food intake patterns and reduces feeding efficiency under restriction.
  • Opioid kappa receptor pathways involved in food intake regulation are fundamentally intact in MSG-treated mice.

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