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Immunopathological study of spleen during Japanese encephalitis virus infection in mice

A Mathur1, M Bharadwaj, R Kulshreshtha

  • 1Postgraduate Department of Microbiology, K.G. Medical College, Lucknow, India.

Insights

Japanese encephalitis virus (JEV) infects macrophages and T-cells in mice, causing spleen changes. Germinal centers appear later, aiding in virus clearance.

Area of Science:

  • Virology
  • Immunology
  • Pathology

Background:

  • Japanese encephalitis virus (JEV) is a significant cause of viral encephalitis globally.
  • Understanding the early pathogenesis and immune response to JEV is crucial for developing effective treatments.

Purpose of the Study:

  • To investigate the early replication sites and splenic immune response following intraperitoneal inoculation of Japanese encephalitis virus.
  • To elucidate the role of splenic cellular changes, including germinal center formation, in JEV clearance.

Main Methods:

  • Indirect immunofluorescence was used to detect JEV replication in splenic macrophages and T-cells.
  • Morphological studies of spleen tissues were conducted at various time points post-infection.
  • Cellular changes, including macrophage proliferation and leukocyte accumulation, were analyzed.

Main Results:

  • JEV replicated in peritoneal macrophages and subsequently in splenic macrophages and T-cells (in PALS).
  • Splenic morphological changes included macrophage proliferation, polymorphonuclear leukocyte accumulation, and enlargement of T-dependent areas.
  • Germinal centers emerged in T-independent areas by day 15, coinciding with the disappearance of virus antigen-containing cells.

Conclusions:

  • JEV infection initially targets macrophages and T-cells within the spleen.
  • The spleen undergoes significant proliferative and inflammatory changes during JEV infection.
  • Germinal center formation appears to play a role in the clearance of Japanese encephalitis virus from the spleen.

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