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Correlation of hippocampal atrophy with hyperhomocysteinemia in hemodialysis patients: An exploratory pilot study

Kyoko Maesato1, Takayasu Ohtake1, Yasuhiro Mochida1

  • 1Department of Kidney Disease and Transplant Center, Shonan Kamakura General Hospital Kamakura, Kanagawa, Japan.

Plos One
|April 11, 2017
PubMed

Insights

Cognitive impairment is a concern for hemodialysis (HD) patients. This study found hyperhomocysteinemia is linked to hippocampal atrophy in HD patients, while age impacts whole-brain atrophy.

Area of Science:

  • Nephrology
  • Neurology
  • Radiology

Background:

  • Cognitive impairment is a significant issue for patients undergoing hemodialysis (HD).
  • The specific factors contributing to brain atrophy in HD patients remain unclear.
  • Understanding these factors is crucial for managing neurological complications in HD patients.

Purpose of the Study:

  • To investigate the association between brain atrophy and various clinical factors in HD outpatients.
  • To quantitatively assess hippocampal and whole-brain atrophy using MRI.
  • To identify independent determinants of brain atrophy in this population.

Main Methods:

  • Brain MRI scans were acquired for 34 HD outpatients.
  • Voxel-based Specific Regional Analysis System for Alzheimer's Disease (VSRAD) software was used for quantitative atrophy assessment.
  • Correlation and multiple regression analyses were performed to identify contributing factors.

Main Results:

  • The mean extent of hippocampal atrophy was 27.3% and whole-brain atrophy was 11.2%.
  • Hippocampal atrophy correlated with low BMI, high total serum homocysteine (tHcy), and elevated brachial-ankle pulse wave velocity (baPWV).
  • Age and hypoalbuminemia were associated with whole-brain atrophy, with age being an independent determinant. Hyperhomocysteinemia (tHcy) was an independent determinant of hippocampal atrophy.

Conclusions:

  • Hyperhomocysteinemia is significantly associated with hippocampal atrophy in hemodialysis patients.
  • Age is a key determinant of whole-brain atrophy in this cohort.
  • Further research is warranted to explore interventions targeting these factors.
Abstract

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