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Significant association of RNF213 p.R4810K, a moyamoya susceptibility variant, with coronary artery disease

Takaaki Morimoto1,2, Yohei Mineharu1, Koh Ono3

  • 1Department of Neurosurgery, Kyoto University Graduate School of Medicine, Kyoto, Japan.

Plos One
|April 18, 2017
PubMed

Insights

The RNF213 p.R4810K variant is linked to coronary artery disease in Japanese individuals. This finding may help understand genetic risk factors for heart disease in Asian populations.

Area of Science:

  • Genetics
  • Cardiovascular Science
  • Neurology

Background:

  • The genetic basis of coronary artery disease (CAD) remains incompletely understood, particularly in Asian populations.
  • Moyamoya disease, a progressive cerebrovascular condition, is sometimes associated with CAD.
  • The RNF213 p.R4810K variant is prevalent in Japanese Moyamoya disease patients, suggesting its potential role in CAD.

Purpose of the Study:

  • To investigate the association between the RNF213 p.R4810K variant and coronary artery disease in the Japanese population.
  • To determine if RNF213 p.R4810K is a risk factor for CAD.

Main Methods:

  • Genotyping of the RNF213 p.R4810K variant in 956 CAD patients and 716 controls.
  • Replication analysis in an independent cohort (311 CAD patients, 494 controls).
  • Multivariate logistic regression analysis, adjusting for known risk factors like dyslipidemia and smoking.

Main Results:

  • The RNF213 p.R4810K minor allele frequency was significantly higher in CAD patients (2.04%) versus controls (0.98%) in the primary study (OR=2.11, p=0.017).
  • After adjusting for risk factors, the association remained significant under a dominant model (OR=2.90, p=0.005).
  • The replication study showed a significant association after adjusting for age and sex (OR=4.99, p=0.031), but not when further adjusted for risk factors (OR=3.82, p=0.076).

Conclusions:

  • The RNF213 p.R4810K variant shows a significant association with coronary artery disease in the Japanese population.
  • This variant may represent a genetic risk factor for CAD in this demographic.
Abstract

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