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Mutations affecting the synthesis of NADP-dependent glutamate dehydrogenase in Pseudomonas aeruginosa

C L Joannou1, P R Brown, R Tata

  • 1Department of Biochemistry, King's College London, UK.

Insights

NADP-dependent glutamate dehydrogenase (NADP-GDH) is not essential for Pseudomonas aeruginosa growth. Glutamate synthase is required for growth when NADP-GDH is absent, indicating its crucial role.

Area of Science:

  • Microbiology
  • Biochemistry
  • Enzymology

Background:

  • Pseudomonas aeruginosa utilizes NADP-dependent glutamate dehydrogenase (NADP-GDH) for amino acid biosynthesis.
  • The essentiality of NADP-GDH for growth and its relationship with glutamate synthase in P. aeruginosa were not fully understood.

Purpose of the Study:

  • To investigate the role of NADP-GDH in Pseudomonas aeruginosa growth.
  • To determine if NADP-GDH is essential for the organism's survival.
  • To understand the interplay between NADP-GDH and glutamate synthase.

Main Methods:

  • Purification of NADP-GDH from Pseudomonas aeruginosa strain 8602.
  • Characterization of purified NADP-GDH using Sephadex gel filtration and SDS-PAGE.
  • Construction and analysis of mutant strains lacking NADP-GDH and/or glutamate synthase.
  • Transduction experiments to map mutations affecting NADP-GDH activity.

Main Results:

  • NADP-GDH was purified, with a molecular weight of 280,000 and subunit molecular weight of 45,000.
  • Mutants lacking both NADP-GDH and glutamate synthase required glutamate for growth.
  • Transductants lacking only NADP-GDH exhibited wild-type growth properties.
  • A mutant with high NADP-GDH activity synthesized normal enzyme and maintained similar intracellular glutamate levels.

Conclusions:

  • NADP-dependent glutamate dehydrogenase is not essential for the growth of wild-type Pseudomonas aeruginosa.
  • Glutamate formation via NAD-dependent glutamate dehydrogenase is not significant in this organism.
  • Glutamate synthase plays a critical role in glutamate biosynthesis when NADP-GDH is absent.
  • The mutation leading to high NADP-GDH expression is linked to the gdhA structural gene.

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