Acute tumor lysis syndrome in non-Hodgkin lymphoma induced by dexamethasone
Related Experiment Video
Updated: Oct 23, 2026
10:52
Tumor Engraftment in a Xenograft Mouse Model of Human Mantle Cell Lymphoma
Published on: March 30, 2018
1Grady Memorial Hospital, Emory University School of Medicine, Atlanta, GA 30303.
American Journal of Hematology
|October 1, 1988
Abstract:
Acute tumor lysis syndrome developed in a 30-year-old man with non-Hodgkin lymphoma after dexamethasone administration. To the best of our knowledge, this case is the first one reported following single agent corticosteroid treatment.
More Related Videos
Related Concept Videos
01:28Graves' Disease I: Introduction
Graves' disease is an autoimmune disorder that causes hyperthyroidism, or overactivity of the thyroid gland. It results from autoantibodies called thyroid-stimulating immunoglobulins (TSIs), which bind to thyroid-stimulating hormone (TSH) receptors, leading to overstimulation of hormone production and a hypermetabolic state.EtiologyAlthough considered idiopathic, Graves’ disease has well-established contributing factors. There is a strong genetic component, with increased prevalence in...
01:24Graves Disease II: Pathophysiology
Graves’ disease is an autoimmune disorder characterized by the production of thyroid-stimulating immunoglobulins (TSI) that activate TSH receptors, leading to excessive synthesis and release of thyroid hormones (T3 and T4) and resulting in hyperthyroidism.Among all causes of hyperthyroidism, Graves’ disease is the most common and can happen at any age, though it is more frequent in women. It produces a hypermetabolic state with features such as weight loss, tachycardia, tremor, and heat...