Redo coronary bypass grafting for congenital left main coronary atresia: a case report

Shin Yajima1, Koichi Toda2, Hiroyuki Nishi2

  • 1Department of Cardiovascular Surgery, Osaka University Graduate School of Medicine, 2-15 Yamadaoka, Suita, Osaka, 565-0871, Japan. s-yajima@surg1.med.osaka-u.ac.jp.

Insights

Congenital left main coronary atresia, a rare condition, was successfully treated with redo coronary artery bypass grafting 27 years after initial surgery. This demonstrates long-term effectiveness of grafts for coronary artery disease management.

Area of Science:

  • Cardiovascular Surgery
  • Pediatric Cardiology
  • Interventional Cardiology

Background:

  • Congenital left main coronary atresia is an exceptionally rare coronary anomaly.
  • Long-term outcomes and optimal management for recurrent ischemia post-surgery are not well-established.
  • This case highlights successful redo coronary artery bypass grafting (CABG) for unstable angina 27 years after initial CABG for congenital left main coronary atresia.

Observation:

  • A 33-year-old woman presented with unstable angina.
  • She previously underwent CABG at age 6 for congenital left main coronary atresia.
  • Despite patent grafts, ischemia persisted due to graft flow disruption.

Findings:

  • Redo CABG using the right internal thoracic artery to the first diagonal branch successfully revascularized the left anterior descending artery territory.
  • The patient experienced complete symptom resolution and remained well for over 3 years post-procedure.
  • Coronary revascularization with saphenous vein and left internal thoracic artery grafts proved effective for long-term distal coronary artery supply.

Implications:

  • This case underscores the potential for long-term graft patency and effectiveness in managing congenital coronary anomalies.
  • It emphasizes the necessity of vigilant long-term surveillance for potential late graft failure or new coronary artery occlusions.
  • Redo CABG can be a viable option for managing recurrent ischemia in adults with complex congenital coronary anomalies.
Abstract

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