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Pathogenesis of acute and persistent murine herpesvirus infection in mice

Acta Virologica
|January 1, 1985
PubMed

Insights

Murine herpesvirus-68 (MHV-68) infection in young mice causes severe pneumonia and spreads to organs. Surviving mice show dynamic MHV-68 persistence, not static latency.

Area of Science:

  • Virology
  • Immunology
  • Pathology

Background:

  • Murine herpesvirus isolate 68 (MHV-68) is a significant pathogen in laboratory mice.
  • Understanding MHV-68 pathogenesis and persistence is crucial for controlling viral infections.

Purpose of the Study:

  • To investigate the pathogenesis and persistence of MHV-68 in outbred mice inoculated at different ages and via different routes.
  • To determine the viral dissemination patterns and long-term viral presence.

Main Methods:

  • Mice were inoculated with MHV-68 at 5, 10, and 21 days old via oral and/or intranasal routes.
  • Pathological examination, immunofluorescence (IF), and electron microscopy were used to detect viral antigen and particles.
  • Virus recovery from surviving mice and their mothers was performed using tissue homogenates and explantation techniques.

Main Results:

  • Infection in 5- and 10-day-old mice led to severe exudative pneumonia and hematogenous dissemination to liver, heart, and kidneys.
  • Viral antigen was detected in various tissues, including lungs, heart, spleen, thymus, kidneys, and Gasserian ganglia.
  • Electron microscopy confirmed virus transfer across capillary endothelium.
  • MHV-68 was recovered from surviving mice and mothers up to 169 days post-infection, indicating dynamic persistence.

Conclusions:

  • Early-life MHV-68 infection causes severe disease and widespread dissemination.
  • The findings suggest a dynamic persistence of MHV-68 rather than a static latent infection in mice.

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