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Purification and characterization of a calmodulin-sensitive adenylate cyclase from Bordetella pertussis

Biochemistry
|November 5, 1985
PubMed

Insights

Bordetella pertussis adenylate cyclase is crucial for whooping cough pathogenesis. Extensive purification revealed its calmodulin sensitivity and high turnover rate, suggesting a key role in disease development.

Area of Science:

  • Microbiology
  • Biochemistry

Background:

  • Bordetella pertussis causes whooping cough and releases adenylate cyclase.
  • This enzyme is essential for B. pertussis virulence.
  • The enzyme's role in increasing intracellular cAMP levels in host cells is proposed.

Purpose of the Study:

  • To purify and characterize the Bordetella pertussis adenylate cyclase.
  • To investigate the enzyme's properties, including calmodulin sensitivity and molecular weight.

Main Methods:

  • Extensive purification of the enzyme from bacterial culture medium.
  • Anion-exchange chromatography and gel filtration chromatography were employed.
  • Enzyme activity, molecular weight, and kinetic parameters were determined.

Main Results:

  • The adenylate cyclase was purified 1600-fold with a specific activity of 608 μmol cAMP min⁻¹ mg⁻¹.
  • The enzyme's molecular weight was 43,400 Da without calmodulin and 54,200 Da with calmodulin.
  • A high turnover number of 27,000 min⁻¹ was observed, the highest documented for any adenylate cyclase.

Conclusions:

  • Bordetella pertussis adenylate cyclase is a potent enzyme contributing to whooping cough pathogenesis.
  • Calmodulin binding significantly alters the enzyme's molecular weight.
  • The enzyme's high catalytic efficiency supports its proposed role in disease.

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