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Published on: February 10, 2013
Regadenoson cardiac stress test-induced stress cardiomyopathy
Jagadeesh K Kalavakunta1, Dominika M Zoltowska2, Yashwant Agrawal2
1Cardiology, Borgess Medical Center/Michigan State University, Kalamazoo, Michigan, USA.
Insights
Takotsubo cardiomyopathy, or stress-induced cardiomyopathy, mimics heart attacks. This case report details a patient who developed stress-induced cardiomyopathy after a cardiac stress test, but recovered with treatment.
Area of Science:
- Cardiology
- Cardiovascular Medicine
- Clinical Case Reports
Background:
- Takotsubo cardiomyopathy (TTC), also known as apical ballooning syndrome or stress-induced cardiomyopathy, mimics acute coronary syndromes.
- It is typically triggered by significant physiological or emotional stress.
Observation:
- A 55-year-old woman presented with congestive heart failure shortly after a regadenoson cardiac stress test.
- Transthoracic echocardiogram showed reduced heart function.
Findings:
- Cardiac catheterization revealed normal coronary arteries.
- Left ventriculography confirmed the diagnosis of apical ballooning syndrome.
- Guideline-directed therapy led to improved heart function and symptom resolution.
Implications:
- This case highlights regadenoson cardiac stress testing as a potential trigger for Takotsubo cardiomyopathy.
- Early recognition and appropriate management are crucial for recovery.
- Further research into stress-induced cardiomyopathy triggers is warranted.
Abstract:
Takotsubo cardiomyopathy, also described as apical ballooning syndrome/stress-induced cardiomyopathy, imitates acute coronary syndrome and is usually related to a massive physiological or emotional stressor. We describe perhaps the first reported case to the best of our knowledge of a 55-year-old Caucasian woman who presented with congestive heart failure after having a regadenoson cardiac stress test a few hours prior to presentation to the hospital. Transthoracic echocardiogram revealed reduced heart function. She had normal coronaries on cardiac catheterisation, and left ventriculography confirmed apical ballooning syndrome. She underwent guideline-directed therapy, and heart function improved in the repeat echocardiogram along with clinical resolution of symptoms.
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