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Gastrointestinal Motility Disorders01:20

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Gastrointestinal or GI motility disorders are characterized by irregular gastrointestinal tract movements, disrupting food transit from the mouth to the anus. They are caused by damage or dysfunction in gut muscles or nerves. These disorders can cause symptoms such as severe constipation, diarrhea, abdominal pain, and swallowing difficulties. Disorders can affect any segment of the GI tract and range widely in severity, from common conditions like GERD to life-threatening conditions like...
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Chronic bowel diseases are a group of long-term conditions affecting the digestive tract, characterized by inflammation and damage to the gut lining. These conditions primarily include irritable bowel syndrome and inflammatory bowel disease.
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Enteric Nervous System: Regulation of GI Motor Activity01:11

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The Enteric Nervous System (ENS) plays a pivotal role in regulating gastrointestinal or GI motor activity. This complex network of nerves, deeply embedded within the gut wall, responds to changes in the gut environment and receives input from both the autonomic nervous system and the central nervous system. By doing so, the ENS operates various programs tailored to the body's nutritional status and needs.
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The neuronal supply to the gastrointestinal (GI) tract is essential for regulating various functions, including digestion, absorption, and movement of food. This intricate network of nerves is known as the enteric nervous system (ENS), often referred to as the "second brain" of the body.
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Crohn's disease
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Irritable Bowel Syndrome I: Introduction01:17

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Irritable Bowel Syndrome (IBS) is characterized by functional disturbances in the gastrointestinal system, presenting a cluster of symptoms without evident structural or biochemical abnormalities. It primarily affects the large intestine and may cause abdominal pain, bloating, excessive gas, diarrhea, constipation, or both.
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Nerve Ultrasound Protocol to Detect Dysimmune Neuropathies
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[Polyneuropathies in intestinal diseases].

I V Damulin1, D A Degterev2

  • 1Sechenov First Moscow State Medical University, Moscow, Russia; Moscow Clinical Research Center, Moscow, Russia.

Zhurnal Nevrologii I Psikhiatrii Imeni S.S. Korsakova
|September 9, 2017
PubMed
Summary
This summary is machine-generated.

Intestinal diseases can cause vitamin B1 (thiamine) and B12 deficiencies, leading to neurological issues like polyneuropathies (PN). Gluten intolerance is a common cause of PN in patients with intestinal problems.

Keywords:
celiac diseasediagnosisglutenpathogenesispolyneuropathytreatmentvitamin deficit

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Area of Science:

  • Neurology
  • Gastroenterology
  • Nutritional Science

Background:

  • Intestinal diseases can lead to malabsorption of essential nutrients, including B vitamins.
  • This malabsorption can cause neurological disturbances, such as polyneuropathies (PN).
  • Factors like gluten sensitivity and inflammation of the stomach and intestinal lining contribute to these neurological issues.

Purpose of the Study:

  • To examine the link between intestinal diseases and vitamin B deficiencies.
  • To explore the neurological consequences, particularly polyneuropathies, arising from these deficiencies.
  • To analyze diagnostic methods for these vitamin-deficiency-related neurological disorders.

Main Methods:

  • Review of existing literature on polyneuropathies and intestinal diseases.
  • Analysis of clinical and paraclinical diagnostic approaches.
  • Correlation of specific vitamin deficiencies (B1, B12) with neurological symptoms.

Main Results:

  • Vitamin B12 deficiency is linked to myelopathy and PN, especially in pernicious anemia.
  • Vitamin B1 (thiamine) deficiency is prevalent in alcoholism and also observed in various gastrointestinal conditions.
  • Both demyelinating and axonal PN can occur in enteropathy patients, with gluten intolerance being a frequent cause.

Conclusions:

  • Intestinal diseases significantly contribute to B vitamin deficiencies, impacting neurological health.
  • Early diagnosis and management of vitamin deficiencies are crucial for preventing or treating PN.
  • Further research into clinical and paraclinical diagnostic tools is warranted.