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The mumps-specific T cell response in healthy individuals and insulin-dependent diabetics: preferential restriction
O Bruserud1, G Paulsen, E Thorsby
1Institute of Transplantation Immunology, University of Oslo, Norway.
Abstract:
An increased frequency of mumps-specific T lymphocytes restricted by DR4-associated elements has previously been described in DR4 heterozygotes. Here we report that the preferential restriction elements may be present on DR4 molecules expressing either Dw4 or Dw14. No particular genomic DQ-beta-polymorphism was associated with the preferential restriction elements.
Insights
Mumps-specific T cells in DR4 heterozygotes are preferentially restricted by Dw4 or Dw14 elements on DR4 molecules. This finding clarifies T-cell restriction in immune responses to mumps virus.
Area of Science:
- Immunology
- Human Genetics
Background:
- Previous studies identified increased mumps-specific T lymphocytes restricted by Human Leukocyte Antigen (HLA)-DR4-associated elements in DR4 heterozygotes.
- The specific DR4 subtypes responsible for this preferential restriction were not fully elucidated.
Purpose of the Study:
- To investigate the specific DR4 subtypes that preferentially restrict mumps-specific T lymphocytes in DR4 heterozygotes.
- To determine if genomic DQ-beta-polymorphism is associated with this preferential restriction.
Main Methods:
- Analysis of T-lymphocyte restriction elements in DR4 heterozygotes.
- Subtyping of DR4 molecules, including Dw4 and Dw14.
- Examination of genomic DQ-beta-polymorphism.
Main Results:
- The preferential restriction of mumps-specific T lymphocytes was found to be associated with DR4 molecules expressing either Dw4 or Dw14 subtypes.
- No association was observed between preferential restriction elements and specific genomic DQ-beta-polymorphisms.
Conclusions:
- Specific DR4 subtypes, namely Dw4 and Dw14, play a key role in the preferential restriction of mumps-specific T lymphocytes.
- Immune response modulation by specific HLA alleles is further clarified, with no apparent influence from DQ-beta-polymorphism in this context.