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Fasciclin-calcareous corpuscle binary complex mediated protein-protein interactions in Taenia solium metacestode

Chun-Seob Ahn1, Jeong-Geun Kim1, Young-An Bae2

  • 1Department of Molecular Parasitology, Samsung Medical Center, Sungkyunkwan University School of Medicine, Suwon, 16419, South Korea.

Parasites & Vectors
|September 22, 2017
PubMed
Abstract

Insights

Taenia solium metacestode Fasciclin (TsMFas) proteins bind to calcareous corpuscles (CC), forming complexes. This interaction involves key metabolic and motility proteins, aiding parasite survival.

Area of Science:

  • Parasitology
  • Molecular Biology
  • Biochemistry

Background:

  • Neurocysticercosis (NC), caused by Taenia solium metacestode (TsM), is a significant global health issue.
  • TsM survival involves diverse bioactive molecules, including Fasciclin (Fas) proteins, which mediate cell adhesion and migration.

Purpose of the Study:

  • To investigate the hypothesis that TsMFas binds to calcareous corpuscles (CC) via adhesive properties.
  • To explore the role of TsMFas-CC interactions in protein-protein interactions and the formation of a symbiotic network for TsM survival.

Main Methods:

  • Isolation and characterization of two paralogous TsMFas proteins (TsMFas1 and TsMFas2).
  • Determination of co-localization patterns of TsMFas with CC.
  • In vitro binding assays and proteomic analysis to identify binding partners and establish protein-protein interaction networks.

Main Results:

  • Two TsMFas proteins (TsMFas1 and TsMFas2) were identified, showing conserved Fas domains and adhesion activity.
  • TsMFas proteins co-localized with CC in TsM tissues.
  • Proteomic analysis revealed seven protein ligands involved in carbohydrate metabolism and cytoskeleton/cellular motility, forming a complex network.

Conclusions:

  • TsMFas and CC likely form a symbiotic interaction.
  • This interaction mediates protein-protein interactions crucial for maintaining homeostasis and prolonged TsM survival within the host.

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