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Potiskum virus: enhancement of replication in a macrophage-like cell line

A H Fagbami1, S B Halstead, N Marchette

  • 1Department of Virology, College of Medicine, University of Ibadan, Nigeria.

Acta Virologica
|November 1, 1987
PubMed

Insights

Potiskum virus replication in macrophage cells was enhanced by specific antiviral antibodies. This antibody-mediated enhancement was most pronounced at lower multiplicities of infection (MOI).

Area of Science:

  • Virology
  • Immunology
  • Cell Biology

Background:

  • Potiskum virus is a pathogen that infects macrophage-like cell lines.
  • Antiviral antibodies can modulate viral replication.
  • Understanding virus-antibody interactions is crucial for developing effective antiviral strategies.

Purpose of the Study:

  • To investigate the effect of subneutralizing concentrations of specific antiviral antibody on Potiskum virus replication.
  • To determine the influence of multiplicity of infection (MOI) on antibody-mediated enhancement of viral replication.

Main Methods:

  • Potiskum virus replication was studied in P388D1 macrophage-like cells.
  • Cells were infected at various multiplicities of infection (MOI) ranging from 0.4 to 0.0004.
  • The effect of specific antiviral antibody (immune mouse ascitic fluid - IMAF) at different dilutions was assessed.

Main Results:

  • Potiskum virus replicated to high titers across all tested MOI.
  • Supplementation with specific antiviral antibody significantly enhanced virus replication.
  • The enhancement of replication was dependent on MOI, with the greatest effect observed at the lowest MOI.
  • The highest enhancement ratio was observed at a 1:500 dilution of IMAF, with an enhancing antibody titre of 5,000.

Conclusions:

  • Subneutralizing concentrations of specific antiviral antibodies can enhance Potiskum virus replication in macrophage-like cells.
  • This enhancement is dependent on the multiplicity of infection, being more significant at lower MOI.
  • These findings highlight a complex interplay between Potiskum virus, host cells, and the immune response, potentially impacting viral pathogenesis and control strategies.

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