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Optic Nerve Transection: A Model of Adult Neuron Apoptosis in the Central Nervous System

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Published on: May 12, 2011

Abducens nerve palsy after orbital decompression.

Shani Golan¹, Robert A Goldberg¹

¹a Department of Ophthalmology , University of California Los Angeles , Los Angeles , California , USA.

Orbit (Amsterdam, Netherlands)

|October 24, 2017

Summary

This summary is machine-generated.

New double vision after thyroid eye disease surgery can occur. Two cases suggest Abducens nerve palsy may result from ischemic neuropathy following reoperative orbital decompression.

Keywords:

Thyroid eye disease abducens nerve orbital decompression

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Area of Science:

Ophthalmology
Neurology

Background:

Thyroid Eye Disease (TED) can cause double vision after orbital decompression surgery, with varying incidence.
Mechanisms for new-onset diplopia post-decompression remain unclear, with theories including muscle fibrosis, cone displacement, or TED reactivation.

Observation:

Two patients with inactive TED underwent uncomplicated secondary orbital decompression.
Postoperatively, both patients developed double vision and limited abduction in the operated eye.

Findings:

Abducens nerve palsy was diagnosed in both patients after secondary orbital decompression.
MRI scans showed no abnormal bleeding or scarring, ruling out these as direct causes.

Implications:

These cases suggest ischemic neuropathy, potentially from subtle hemorrhage or inflammation, as a cause of Abducens palsy after reoperative orbital decompression.
Understanding these mechanisms is crucial for managing diplopia in TED patients undergoing repeat surgeries.