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Updated: Feb 14, 2026

Acute Myocardial Infarction in Rats
Published on: February 16, 2011
Accelerated idioventricular rhythm degenerating into bidirectional ventricular tachycardia following acute myocardial
Yun-Tao Zhao1, Hang Zhou1, Yumin Cui2
1Department of Cardiology, Aerospace Center Hospital, 15 Yuquan road, Beijing, 100049, People's Republic of China.
Abstract:
Bidirectional ventricular tachycardia (BVT) is a rare ventricular tachyarrhythmia. It is usually regular, demonstrating a beat-to-beat alternation in the QRS frontal axis that varies between -20° to -30° and +110°. The tachycardia rate is typically between 140 and 180 beats/min and the QRS is relatively narrow, with a duration of 120 to 150 ms. The etiology of published BVT cases is most commonly digitalis toxicity and, rarely, herbal aconitine poisoning, hypokalemic periodic paralysis, catecholaminergic polymorphic ventricular tachycardia (CPVT), myocarditis, and Andersen-Tawil syndrome. We report a case of accelerated idioventricular rhythm (AIVR) degenerating into BVT following acute myocardial infarction, and briefly discuss the proposed mechanisms underlying BVT.
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