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Peri-implantitis.

Frank Schwarz1, Jan Derks2, Alberto Monje3,4

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|June 22, 2018
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Summary
This summary is machine-generated.

Peri-implantitis involves inflammation and bone loss around dental implants. Key risk factors include prior periodontitis and poor oral hygiene, though smoking and diabetes links remain unclear.

Keywords:
diagnosisimplantologyperi-implantitissystematic reviews and evidence-based medicine

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Area of Science:

  • Dental Implantology
  • Periodontology
  • Oral Pathology

Background:

  • Peri-implantitis is a significant complication following dental implant placement.
  • Understanding its definition, progression, and risk factors is crucial for effective management.
  • The 2017 World Workshop provided updated classifications for periodontal and peri-implant diseases.

Purpose of the Study:

  • To provide an evidence-based overview of peri-implantitis.
  • To review the definition, conversion from mucositis, disease progression, characteristics, and risk factors.
  • To discuss progressive bone loss without soft tissue inflammation.

Main Methods:

  • A narrative review of existing literature.
  • Focused on topics including peri-implantitis definition, progression, and risk indicators.
  • Examined crestal bone loss in the absence of soft tissue inflammation.

Main Results:

  • Peri-implantitis is defined by peri-implant connective tissue inflammation and bone loss.
  • Disease onset can be early, with non-linear, accelerating progression.
  • Increased probing depths and inflammatory lesions are characteristic; bone loss is often circumferential.
  • History of periodontitis and poor plaque control are strong risk factors; smoking/diabetes data are inconclusive.
  • Submucosal cement and lack of keratinized mucosa show limited evidence as risk factors.

Conclusions:

  • Peri-implantitis presents with inflammation and progressive bone loss around implants.
  • Risk is elevated in patients with prior periodontitis and inadequate maintenance.
  • Further research is needed to clarify the role of certain risk factors and the progression of bone loss without soft tissue inflammation.