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Related Experiment Video

Updated: Feb 7, 2026

Investigation of Beige Fat Biology and Metabolism Using the CRISPR SunTag-p65-HSF1 Activation System
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Common traits between the beige fat-inducing stimuli.

Ozren Stojanović1, Silas Kieser1, Mirko Trajkovski2

  • 1University of Geneva, Faculty of Medicine, Department of Cell Physiology and Metabolism, Centre Médical Universitaire, 1211 Geneva, Switzerland; University of Geneva, Diabetes Centre, Faculty of Medicine, 1211 Geneva, Switzerland.

Current Opinion in Cell Biology
|July 15, 2018
PubMed
Summary

Adipose tissue browning, or increased energy expenditure, is influenced by gut microbiota and negative energy balance. These factors modulate adipocyte function, impacting metabolic homeostasis and obesity.

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Area of Science:

  • Metabolic Physiology
  • Adipose Tissue Biology
  • Microbiome Research

Background:

  • Adipose tissue is crucial for metabolic homeostasis, but excess fat (obesity) causes disorders.
  • Adipocytes exhibit plasticity, shifting between energy storage and expenditure (browning).
  • Adipose browning is regulated by tissue environment, sympathetic nerves, and immune responses.

Purpose of the Study:

  • To review recent findings on how metabolic stimuli influence adipocyte function.
  • To highlight the roles of gut microbiota and negative energy balance in regulating adipose browning.

Main Methods:

  • Literature review of metabolic stimuli impacting adipocyte function.
  • Focus on studies investigating gut microbiota and energy balance effects on browning.

Main Results:

  • Metabolic stimuli significantly shape adipocyte plasticity and function.
  • Gut microbiota and negative energy balance are key regulators of adipose tissue browning.
  • Sympathetic innervation and immune responses integrate signals promoting browning.

Conclusions:

  • Understanding adipocyte plasticity is vital for metabolic health.
  • Targeting gut microbiota and energy balance may offer strategies to combat obesity and related disorders.