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Herpes simplex virus (HSV) spreads through lymphatic vessels to sensory ganglia, particularly in younger mice. Lymphatic dissemination may play a role in HSV

Area of Science:

  • Virology
  • Immunology
  • Neuroscience

Background:

  • Herpes simplex virus (HSV) establishes lifelong infections.
  • Understanding HSV dissemination is crucial for developing effective antiviral strategies.
  • Previous research has focused on neuronal pathways, but lymphatic spread is less understood.

Purpose of the Study:

  • To investigate the role of lymphatic spread in the dissemination of herpes simplex virus (HSV) in mice.
  • To determine the primary sites of HSV replication and spread following peripheral inoculation.
  • To compare HSV dissemination patterns in young versus older mice.

Main Methods:

  • Percutaneous inoculation of HSV into the footpad or orofacial area of mice.
  • Virus isolation from various lymph nodes (popliteal, inguinal, axillary, lumbar, submandibular) at different time points.
  • Detection of virus in sensory ganglia (ipsilateral and contralateral).
  • Comparison of results between young and older mice.

Main Results:

  • HSV was frequently detected in draining lymph nodes (popliteal, submandibular) after inoculation.
  • Contralateral lumbar lymph nodes showed frequent HSV detection after footpad inoculation.
  • HSV presence in lymph nodes correlated with sensory ganglia invasion.
  • HSV dissemination to contralateral sensory ganglia was more pronounced in younger mice.
  • Older mice showed less frequent lymph node detection and limited contralateral ganglia spread.

Conclusions:

  • Lymphatic spread is a significant route for HSV dissemination to lymph nodes and potentially to contralateral sensory ganglia.
  • Age influences HSV dissemination patterns, with younger mice exhibiting more extensive spread.
  • These findings highlight the importance of the lymphatic system in HSV pathogenesis and suggest potential therapeutic targets.

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