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Rat Model of Photochemically-Induced Posterior Ischemic Optic Neuropathy
Published on: November 29, 2015
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Surgical Treatment for Traumatic Optic Neuropathy
Hyuk-Jin Oh1, Dong-Gyu Yeo2, Sun-Chul Hwang3
1Department of Neurosurgery, Soonchunhyang University Cheonan Hospital, Cheonan, Korea.
Korean Journal of Neurotrauma
|November 8, 2018
Summary
Traumatic optic neuropathy (TON) causes severe vision loss. Surgical decompression of the optic canal (OC) may benefit select patients with bony compression or progressive vision loss, though evidence for high-dose steroids is limited.
Area of Science:
- Ophthalmology
- Neurosurgery
- Trauma Surgery
Background:
- Traumatic optic neuropathy (TON) is a significant cause of vision impairment following head and facial trauma.
- Current treatments like high-dose steroids and surgery lack definitive evidence for improving visual acuity in indirect TON.
Purpose of the Study:
- To review surgical approaches for optic canal (OC) decompression in traumatic optic neuropathy.
- To delineate surgical indications, timing, and techniques for OC decompression.
Main Methods:
- Literature review focusing on surgical interventions for TON.
- Analysis of surgical techniques for optic canal decompression.
Main Results:
- Surgical decompression is recommended for TON with direct bony compression of the optic nerve.
- It may be considered for indirect TON with progressive visual loss.
- Evidence supporting high-dose steroids for TON remains inconclusive.
Conclusions:
- Neurosurgeons must understand the specific indications, optimal timing, and techniques for OC decompression in TON.
- Surgical intervention is a critical consideration for specific TON cases, particularly those involving bony compression.
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