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Spasticity and contracture. Physiologic aspects of formation
M J Botte1, V L Nickel, W H Akeson
1Division of Orthopaedics and Rehabilitation, University of California, San Diego Medical Center 92103.
Clinical Orthopaedics and Related Research
|August 1, 1988
Summary
Upper motor neuron damage causes muscle spasticity, leading to joint stiffness and contractures. Preventing contractures through joint mobilization is crucial for managing spasticity patients.
Area of Science:
- Neurology
- Rehabilitation Medicine
- Neuroscience
Background:
- Disruption of upper motor neuron inhibitory pathways, often due to stroke, brain trauma, or spinal cord injury, results in muscle spasticity.
- Spasticity manifests as increased muscle tone, hyperactive reflexes, and potentially clonus or rigidity.
- This heightened muscle tone can impede joint motion, ultimately leading to the development of contractures.
Purpose of the Study:
- To highlight the challenges in treating established contractures in spasticity.
- To emphasize the importance of preventing contractures in patients with spasticity.
Main Methods:
- This study is a review of existing literature on spasticity and contracture management.
- Focuses on the principles of joint mobilization for contracture prevention.
Main Results:
- Established contractures are difficult to treat effectively.
- Proactive joint mobilization is a key strategy for preventing contractures.
Conclusions:
- Contracture prevention is a primary goal in managing spasticity.
- Early and consistent joint mobilization can mitigate the long-term complications of spasticity.
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