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Thyrotrophin receptor antibody concentration and activity, several years after treatment for Graves' disease

Preethi Nalla1, Stuart Young2, Jane Sanders2

  • 1Section of Endocrinology and Biochemistry, Aneurin Bevan University Health Board, Caerphilly, UK.

Clinical Endocrinology
|November 29, 2018
PubMed
Abstract

Insights

Thyroid-stimulating hormone receptor antibodies (TRAb) decrease after Graves' disease treatment but can persist for years. Surgery most effectively reduced TRAb levels, with most patients showing stimulatory antibody activity.

Area of Science:

  • Endocrinology
  • Immunology
  • Thyroidology

Background:

  • Thyroid-stimulating hormone receptor antibodies (TRAb) are key drivers of autoimmune hyperthyroid diseases like Graves' disease (GD).
  • Monitoring TRAb levels post-treatment is crucial for assessing therapeutic efficacy and disease recurrence risk.

Purpose of the Study:

  • To evaluate the changes in TRAb concentration and activity (stimulating and blocking) after various GD treatments.
  • To determine the long-term persistence of TRAb and its correlation with treatment modalities.

Main Methods:

  • Serum TRAb concentration, stimulating (TSAb), and blocking (TSBAb) activity were measured in 66 patients post-treatment.
  • Patients received carbimazole, radioiodine, or surgery.
  • Thyroid peroxidase antibodies (TPOAb), thyroglobulin antibodies (TgAb), and GAD antibodies (GADAb) were also assessed.

Main Results:

  • TRAb levels decreased across all treatment groups, with surgery showing the most significant reduction.
  • Forty-five percent of patients remained TRAb-positive for over a year, and 23% for over five years.
  • TRAb and TSAb showed a strong correlation (r=0.83), with 70% of TRAb-positive patients having TSAb activity.

Conclusions:

  • While TRAb levels generally decline post-treatment, they can persist long-term in a significant portion of patients.
  • Surgical intervention appears most effective in reducing TRAb concentrations.
  • The predominant activity of TRAb is stimulatory, highlighting the ongoing autoimmune drive in Graves' disease.

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