Removing acesulfame with the peroxone process: Transformation products, pathways and toxicity

Emerging contaminants (ECs) are receiving considerable attention because of their widespread occurrence, persistence and potential threat to the environment, wildlife and humans. Acesulfame (ACE), an extensively used artificial sweetener, is the most worrisome example of ECs. The photolysis/photocatalysis, chlorination and/or permanganate oxidation of ACE produces transformation products (TPs) that are more persistent and toxic than precursors. Thus, an alternative treatment method to treat ACE is required; oxidation by the peroxone process could be that method and was systematically investigated, as reported here. During the peroxone process, ACE degradation followed pseudo-first-order kinetics, with a rate that was significantly higher than after conventional ozonation. The hydroxyl radical was the major reactive species. Amount of hydrogen peroxide (H₂O₂) used, pH and type of water matrix showed significant influence on ACE degradation. Fifteen TPs in ultrapure water extracts, including four newly reported compounds, were identified and characterized by high resolution mass spectrometry (HR-MS) based on accurate mass measurements and MS/MS fragmentation. The reduced toxicity compared to other reported treatments of ACE was likely due to different transformation pathways and TPs generated. The peroxone process therefore appears to be one viable choice for safe removal of ACE.